oalib

Publish in OALib Journal

ISSN: 2333-9721

APC: Only $99

Submit

Any time

2019 ( 25 )

2018 ( 185 )

2017 ( 204 )

2016 ( 260 )

Custom range...

Search Results: 1 - 10 of 144316 matches for " F Zaree "
All listed articles are free for downloading (OA Articles)
Page 1 /144316
Display every page Item
Share of Nations in 37 International Public Health Journals: An Equity and Diversity Perspective Towards Health Research Capacity Building
N Keshavarz Mohammadi,F Zaree,E de Leeuw,MM Emamjomeh
Iranian Journal of Public Health , 2011,
Abstract: Background: This paper contributes to further exploration of inequity in access to health research capacity development by examining the representation of different nations in international public health journals. It also aims to examine the degree of diversity that exists in these journals.Methods: This study is a descriptive survey. It was done with objective sampling on 37 ISI health journals on October of 2008.The number and nationality of people in different editorial positions of the journals was identified. The second analy-sis involved recalculating the numbers obtained for each nation to the population size of nations per million inhabitants. In order to better compare countries in terms of presence in editorial team of the journals, a ‘public health editor equity gap ratio' (PHEEGR) was developed.Results: Low income countries have occupied none of the leadership positions of chief editor or associate /assistant chief editors and middle income countries at maximum shared less than 5 percent. The PHEEGR gap in access to the different editorial positions between highest to the lowest representation of countries was 16/1 for chief editors, 12/1 for associate editors , 335/1 for editorial boards and 202/1 for associate editorial boards. However, after normalizing the data to the country's population, the gap increased significantly.Conclusion: There is an imbalance and possibly even inequity in the composition of editorial boards and offices of interna-tional health journals that should be paid significant attention. This can contribute to fill the equity gap exists between health in developing and developed countries.
ASSOCIATION BETWEEN GLAUCOMA AND BLOOD GROUPS
R. Zaree,Y. Eslami,G. Fakhraie,F. Ghannadi R. Varmazyar
Acta Medica Iranica , 2006,
Abstract: There are reports from different countries that some types of glaucoma are associated with blood groups. This cross-sectional study was performed on 400 glaucomatous patients [100 patients in each group of Primary open angle glaucoma (POAG), chronic angle closure glaucoma (CACG), pseudoexfoliative glaucoma (PEXG) and primary congenital glaucoma (PCG)] and 400 blood donors as control group to assess the association between blood groups and glaucoma. All patients underwent ABO and Rh blood group testing. The prevalence of blood group A was 30% in the control group, 27% in POAG, 33% in CACA, 38% in PEXG and 36% in PCG. The prevalence of blood group B was 24% in the control group, 19% in POAG, 20% in CACG, 15% in PEXG and 34% in PCG (P < 0.025). The prevalence of blood group AB was 8% in the control group, 9% in POAG, 5% in CACG, 12% in PEXG, and 8% in PCG. The prevalence of blood group O was 38% in the control group, 45% in POAC, 42% in CACG, 35% in PEXG and 22% in PCG (P < 0.001). The prevalence of Rh+ was 88% in the control group, 84% in POAG, 87% in CACG, 86% in PEXG and 87% in PCG. Compared to control group, blood group B was more prevalent and blood group O was less prevalent in PCG. There was no association between other types of blood groups (ABO and Rh) and PCG. There was no association between blood groups (ABO and Rh) and other types of glaucoma.
Comparison of the In Vitro Mutagenicity of Artemisia draconculus L. With Sodium Dichromate by Performing Single Cell Gel Electrophoresis (SCGE) or the Comet Assay
Heibatullah Kalantari,Hamid Galehdari,Zahra Zaree,Golnar Varnasery
Jundishapur Journal of Natural Pharmaceutical Products , 2012,
Abstract: Background: The increasing use of herbal drugs and their easy availability have necessitated the use of mutagenicity tests to analyze their toxicity and safety..Objectives: The aim of this study was to determine the in vitro mutagenicity of Artemisia draconculus L., a herbal drug, by performing single cell gel electrophoresis (SCGE)..Materials and Methods: In this study, we obtained a herbal drug with A. draconculus at a density of 0.94; doses of 100 μl, 200 μl, 400 μl, and 800 μl equivalent to 94 mg, 188 mg, 376 mg, and 752 mg of A. draconculus, respectively, were used. Sodium dichromate at a dose of 262 mg was considered to be the positive control, and blood was considered to be the negative control. Blood samples were centrifuged at 3500 rpm for 5 min, and the lower portion of the residue was isolated and mixed with low melting point agarose..Results: A cell suspension was prepared and applied on pre-coated agarose gel slides. Lysis, electrophoresis under alkaline conditions, staining of DNA, comet visualization, and comet scoring were carried out. The statistical analysis of the obtained results showed that with an increase in the dosage of A. draconculus, DNA damage also increased significantly (P < 0.05)..Conclusions: These findings provide valuable information regarding the safety and toxicity of this herbal drug, and this information will be helpful in ensuring rational use of this drug.
Critical role of GSH in Sulfur Mustard-induced Oxidative Stress and Cytotoxicity in Human Skin Fibroblast Cell Line
Ali Beman Zaree Mahmoudabad,Mehdy Saberi,Jilla Pirzad
Iranian Journal of Pharmaceutical Research , 2008,
Abstract: In this study the role of glutathione (GSH) in sulfur mustard -induced oxidative stress and cytotoxicity, in human skin fibroblast cell line (HF2FF) was evaluated. Sulfur mustard-induced superoxide radical and hydrogen peroxide formation were evaluated by determination of superoxide dismutase and catalase activity in cell lysate. The cytotoxicity of sulfur mustard was estimated by lactate dehydrogenase leakage. The intracellular GSH content was modulated by N-acetylcysteine (NAC), a GSH precursor, and buthionine sulfoximine (BSO), a specific GSH synthesis inhibitor. It was found that sulfur mustard exposure led to a dose-and time-dependent decrease in GSH content in HF2FF cells. NAC increased intracellular GSH level and protected the cells against sulfur mustard-induced reactive oxygen species formation and lactate dehydrogenase leakage. In contrast, buthionine sulfoximine pretreatment depleted cellular GSH and enhanced the susceptibility of HF2FF to the cytotoxic effects of sulfur mustard. These results indicated that GSH plays a critical role in protecting HF2FF cell line against sulfur mustar-induced cell injury, most probably through its antioxidant activity.
Cytotoxic and Oxidative Stress Caused by Cadmium and Lead on Human Skin Fibroblast Cells
Ali Beman Zaree Mahmodabady,Mehdi Saberi,Hossein Eimani,Jila Pyrzad
Cell Journal , 2006,
Abstract: Introduction: Heavy metals are important occupational andenvironmental pollutants that cause damage to various organs.Although there is no effective therapy for such a poisoning,metallothionein has been shown to play a key role in thedetoxification of cadmium (Cd). Evidence in the literature suggeststhat superoxide dismutase, glutathione peroxidase, and catalaseconstitute important defense mechanisms against oxygen toxicity inthe cells. The aim of this study was to investigate the effect ofcadmium chloride and Pb-acetate on antioxidant enzymes in thehuman skin fibroblast cells (HF2FF).Material and Methods: The human skin fibroblast (HF2FF) cellswere incubated in serum-free medium containing 20 μM CdCl2 for18 hr three times a week. The same exposure to an equimolar doseof Pb-acetate was performed. After each exposure and after threetimes exposure the cells were collected and cell viability, thecontents of superoxide dismutase (SOD), catalase, glutathioneperoxidase (GSH-Px), GSH and malondialdehyde (MDA) weremeasured.Results: Cd caused cytotoxicity and inhibition of glutathioneperoxidase (GSH-Px) and SOD activity, as well as depletion of thereduced form of glutathione (GSH) in the cell. The level of lipidperoxidation (LP) was increased, but catalase activity was notsignificantly altered. These defects were increased with repeatedexposures. The same exposure to an equimolar dose of Pb-acetateevoked only inhibition of GSH-Px and SOD. The values of GSH,catalase and LP activity remained unchanged.Conclusion: The inhibition of GSH-Px and SOD may be consideredas an important biomarker of the toxic effect of metals.
Excitatory and inhibitory effects of nitric oxide on weight, size, and histological changes of rat cerebellum
Karambaksh A,Noori Mougahi SMH,Hassan Zadeh GR,Tak Zaree N
Tehran University Medical Journal , 2013,
Abstract: Background: Nitric oxide (NO) is produced in different body organs in mammals and numerous physiological and pathological properties are attributed to this small molecule. The precursor of this substance in the body, L-arginine, is synthesized by the enzyme nitric oxide synthase (NOS), and it is catalyzed, and is inhibited by a substance called L-NG-nitroarginine methyl ester (L-NAME). In this study we investigated the qualitative and quantitative effects of nitric oxide on cerebellar histopathology in vivo environment via increasing and decreasing its production.Methods: Forty Wister rats, weighing 200- 250 gr with a mean age of 8 weeks, were divided into 5 groups after making sure the rats were pregnant. Except the control group, the other pregnant groups, respectively received: 2 ml/kg normal saline, 200 mg/kg L-arginine, 20 mg/kg L-NAME and a mixture of the same doses of L-arginine and L-NAME on the third, fourth and fifth days of pregnancy. On day 18 of pregnancy, we anesthetized the rats, excised the cerebellum after craniotomy and fixed the organs in 10% formalin. We later prepared 5 to 6-micron in thickness tissue sections and dyed them by the routine Hematoxylin and eosin (HE) and Masson's Trichrom staining methods before studying them by light microscopy.Results: There was a significant difference between the rats receiving L-arginine and the rats in other groups (P<0.01). Conclusion: This study showed that L-NAME is capable of significantly decreasing the injury caused by nitric oxides in rat cerebellum.
Effect of Acute Toxicity of Cadmium in Mice Kidney Cells
Masoomeh Masoomi Karimi,Moslem Jafari Sani,Ali Zaree Mahmudabadi,Asma Jafari sani
Iranian Journal of Toxicology , 2012,
Abstract: Background: Cadmium is one of the most toxic heavy metals in our environmenthaving a very strong ability to accumulate in body organs, especially in kidney. Thepresent study was done to determine the genotoxicity and cytotoxicity in kidneys ofrats exposed to cadmium.Methods: Male rats (n=30), kept in standard conditions were used in this study.The animals were randomly divided into 2 groups (control and treatment). Thetreatment group was intraperitoneally injected with Cd (300μm/kg) at hours 0, 6,12, 24, 48. Twenty four hours after the last injection, the rats were sacrificed andtheir kidneys were obtained. Then oxidative stress markers, malondialdehide(MDA), glutathione (GSH), and superoxide dismutase (SOD), were assayed inhomogenized kidney for studying their cytotoxicity. For genotoxicity and DNAdamage studies, Comet assay was run on isolated kidney cells. Data analysis wasdone by t-test and ANOVA using SPSS software version 15.Results: MDA and GSH concentrations in normal and Cd exposed kidney cellswere 287.01±37.30nmol/g.pr and 15.61±3.89μmol/g.pr and 609.24±87.87nmol/g.prand 28.52±5.22μmol/g.pr, respectively. In addition, SOD activity in normal and Cdexposed kidney cells were 77.75±4.12 and 218.91±5.40 U/mg.pr, respectively.Comet assay results (content comet length, tail length, and head diameter)showed DNA breakage in the treatment group that was stimulated by Cd whichwas not seen in the control group.Conclusion: The results demonstrated the genotoxicity effect of Cd on kidneycells as well as the ability of Cd to producing cytotoxicity.
Evaluating the Relationship between the Banking System Stability and the Internal Capital Adequacy Assessment Process: Evidence from the Egyptian Banking Sector  [PDF]
Karim F. F. Mohamed
Journal of Financial Risk Management (JFRM) , 2018, DOI: 10.4236/jfrm.2018.74020
Abstract: In the repercussions of the latest financial crisis that have occurred on the years 2008-2009, to fortify the stability of the banking systems, policy makers, and the Basel Committee on Banking Supervision—BCBS, together with national regulators have built up a few safety measures, and structures to guarantee that banks establishments keep up adequate capital levels through using risk management tools, in specific the Internal Capital Adequacy Assessment Processes (ICAAP). They all have called for thorough evaluations and assessments for the structure and components of risk management frameworks, tools, and practices whether by banks, regulators, analysts and risk management experts consistently, to ascertain the adequacy of the banking systems, policies, arrangements and techniques for overseeing risks, and guaranteeing the sufficiency of holding appropriate capital levels for confronting normal, as well as adverse and unexpected situations or emergencies. The main objectives of this research study are to shed the light on the ICAAP as one of the main keys of risk management programs, a process by which banks can use to ensure that they operate with an appropriate level of capital, forward looking processes for capital planning covering a broad range of risks across banks, activities beyond simple capital management, and bring together risk and capital management activities in a form that can be used to support business decisions. The research study shall evaluate the significant relationship between the Banking System Stability (dependent variable) and the Internal Capital Adequacy Assessment Process (ICAAP—independent variable) with evidence from the Egyptian Banking Sector.
Kroniek
- F.
Nieuwe West-Indische Gids , 1949,
Abstract:
Kroniek
- F.
Nieuwe West-Indische Gids , 1949,
Abstract:
Page 1 /144316
Display every page Item


Home
Copyright © 2008-2017 Open Access Library. All rights reserved.