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First, second, and third line medical treatments of vulvodynia are of limited efficacy. Surgical resection, the fourth line treatment of vulvodynia, may have unforgiving sequela. Therefore, acupuncture and electromyographic (EMG) biofeedback could bridge between medical and surgical treatments of vulvodynia. Of note, EMG biofeedback is more frequently recommended in treatment algorithms for vulvodynia than is acupuncture. Trials of acupuncture for unprovoked vulvodynia demonstrate variable efficacy, whereas trials of EMG biofeedback for provoked vulvodynia demonstrate consistent efficacy. Trials of acupuncture for treatment of provoked and unprovoked vulvodynia using identical acupoints, a vulvar algesiometer for objective pain measurement, and standardized, validated, tools for outcome assessment are needed. Such trials may enable comparison of acupuncture to EMG biofeedback for the treatment of provoked and unprovoked vulvodynia. Similarly, trials of EMG biofeedback for treatment of unprovoked vulvodynia would increase the knowledge base of EMG biofeedback for treatment of vulvodynia.
aim of this feasibility study was to contrast rectus abdominis (RA) muscle
strength and electrical activity after two abdominal training protocols, conventional
kinesiotherapy (CK) and Pilates mat exercises (PME). 13 participants
were randomized to one of two groups: CK and PME, and were trained accordingly.
The upper rectus abdominis
(URA) and lower rectus abdominis (LRA) were independently examined and intra-group
and inter-group comparison were done. Findings demonstrate a significant increase
in RA strength after both protocols, relative to baseline. Significantly
increased electrical activation was seen in the URA after CK. Training with
PME, although as efficient as CK in strength improvement, produced decreased
activation level of muscles. CK training induced an opposite result. The
differences after training suggest that CK and PME training programmers may
strength RA, but the neuromuscular activation strategies for that are not the
same. PME can be incorporated into protocols to improve abdominals strength,
and trunk stabilization.
The overall goal of this project is to develop a humane non-human primate model of traumatic spinal cord injury that will facilitate the development and evaluation of therapeutic interventions. The model utilizes neurophysiological techniques to identify the location of the upper motor neuron axons that innervate the lower motor neurons that control tail musculature. This facilitates the placement of a selective lesion that partially disconnects the upper and lower motor neuron supply to the musculature of the tail. An implanted transmitter quantitatively measures electromyography data from the tail. The preliminary data indicates that this model is feasible. The subject was able to tolerate the implantation of the transmitter, without adverse effects. As well, there was no limb impairment, bowel dysfunction or bladder dysfunction. The histopathologic and electromyographic features of the selective experimental lesion were similar to human spinal cord injury.