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Search Results: 1 - 10 of 27641 matches for " Dayi Hu "
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Lessons learned from the TIMI trials in rescue interventions for elderly patients after failed fibrinolytic therapy: look beyond the TIMI flow

Dayi HU,Yifang GUO,Thach Nguyen,

老年心脏病学杂志(英文版) , 2005,
Abstract: In ST-segment elevation myocardial infarction ( STE-MI), acute reperfusion of the infarct-related artery (IRA) is the main goal in the early minutes after the patient seeks medical attention. Fibrinolytic therapy (FT) and/or primary coronary intervention (PCI) were proven to be ef-fective in opening the IHA.
Apolipoprotein E polymorphism in northern Chinese elderly patients with coronary artery disease
Yangchun ZOU,Xingyuan JIA,Xinchun YANG,Liang CUT,Dayi HU,Xiufang HONG,
Yangchun ZOU
,Xingyuan JIA,Xinchun YANG,Liang CUT,Dayi HU,Xiufang HONG

老年心脏病学杂志(英文版) , 2006,
Abstract: Background and objective Apolipoprotein E is a constituent of lipoproteins with considerable variation due to cysteine-argin-ine exchanges. We investigated the relationship between apo E gene polymorphism and the occurrence of coronary artery disease (CAD) in the older population of northern China. Methods The distribution of the Hhal polymorphisms of the apolipoprotein E gene was determined among 55 patients with CAD (CAD group), which was compared with that of 36 elderly subjects without CAD (control group). Results Genotype distributions at both sites (apo E gene 112-bp and 158-bp sites ) were different between the CAD and control groups. The CAD group had lower apolipoprotein E"el"frequencies than the control group (/><0.05). Conclusion Individu-als with apolipoprotein E"e2"are likely to have a reduced risk of developing coronary artery disease as demonstrated by elderly subjects in Northern China.
Elevated serum uric acid level as a predictor for cardiovascular and all-cause mortality in Chinese patients with high cardiovascular risk
Elevated serum uric acid level as a predictor for cardiovascu-lar and all-cause mortality in Chinese patients with high cardiovascular risk

Yongquan Wu,Meijing Li,Jue Li,Yingyi Luo,Yan Xing,Dayi Hu,
Yongquan Wu
,Meijing Li,Jue Li,Yingyi Luo,Yan Xing,Dayi Hu

老年心脏病学杂志(英文版) , 2008,
Abstract: Objective To assess the predictive value of serum uric acid levels for cardiovascular and all-cause mortality in a large prospective population based study.Methods The study was based on 3648 participants in Shanghai and Beijing,who were inpatients with high cardiovascular(CV) risk at baseLine (2004.7 to 2005.1),and blood was taken.Follow-up for death from cardiovascular disease and any cause was complete until January 1,2006.Results The mean follow-up was 1 years.There were 303 deaths during follow-up,of which 121 were cardiovascular.Crude mortality rates were 8.3 % for all patients,6.8% for female patients (116/1715),and 9.7% (187/1933) for male patients.Among men,patients in the lower and higher uric acid groups had increased cardiac and overall mortality risks compared with patients in the normal uric acid groups.Similar relation was found in women but not statistically significant.After adjusting for other conventional risk factors (age,diabetes,hypertension,diuretic use and smoking),baseline uric acid level was still associated with increased risk for death from cardiovascular disease (P=0.005),or death from all causes (P=0.014) Conclusion Our data suggest that abnormal serum uric acid levels are independently and significantly associated with risk of cardiovascular and all-cause mortality.(J Geriatr Cardiol 2008;5:15-20)
Association between High-Sensitivity C-Reactive Protein and N-Terminal Pro-B-Type Natriuretic Peptide in Patients with Hepatitis C Virus Infection
Wenliang Che,Buchun Zhang,Wenling Liu,Yidong Wei,Yawei Xu,Dayi Hu
Mediators of Inflammation , 2012, DOI: 10.1155/2012/730923
Abstract: Background. Prior study showed HCV-infected patients have increased serum N-Terminal Pro-B-Type Natriuretic Peptide (NT-proBNP) and a possible left ventricular diastolic dysfunction. The objectives of the present paper were to investigate the characteristics of hs-CRP and its correlation with clinical profiles including NT-proBNP and echocardiographic variables in HCV-infected patients. Methods and Results. A total of 106 HCV-infected patients and 106 control healthy individuals were enrolled. The level of serum hs-CRP (median 1.023 mg/L, range 0.03~5.379 mg/L) was significantly lower in all 106 patients than that in controls (median 3.147 mg/L, range 0.08~7.36 mg/L, =0.012). Although hs-CRP did not correlate significantly with NT-proBNP when all patients and controls were included (=0.169, =0.121), simple regression analysis demonstrated a statistically significant linear correlation between hs-CRP and NT-proBNP in HCV-infected patients group (=0.392, =0.017). Independent correlates of hs-CRP levels (2=0.13) were older age (=0.031, =0.025) and NT proBNP (=0.024, =0.017). Conclusions. Although the level of serum hs-CRP decreased significantly, there was a significant association between hs-CRP and NT-proBNP in HCV-infected patients.
Decreased Glomerular Filtration Rate Is Associated with Mortality and Cardiovascular Events in Patients with Hypertension: A Prospective Study
Rui Zhang, Liqiang Zheng, Zhaoqing Sun, Xingang Zhang, Jue Li, Dayi Hu, Yingxian Sun
PLOS ONE , 2011, DOI: 10.1371/journal.pone.0027359
Abstract: Background Few studies reported the associations between decreased glomerular filtration rate (GFR) and mortality, coronary heart disease (CHD), and stroke in hypertensive patients. We aim to assess the associations between GFR and mortality, CHD, and stroke in hypertensive patients and to evaluate whether low GFR can improve the prediction of these outcomes in addition to conventional cardiovascular risk factors. Methods and Findings This is an observational prospective study and 3,711 eligible hypertensive patients aged ≥5 years from rural areas of China were used for the present analysis. The associations between eGFR and outcomes, followed by a median of 4.9 years, were evaluated using Cox proportional hazards models adjusting for other potential confounders. Low eGFR was independently associated with risk of all-cause mortality, cardiovascular mortality, and incident stroke [multivariable adjusted hazard ratios (95% confidence intervals) for eGFR <60 ml/min/1.73 m2 relative to eGFR ≥90 ml/min/1.73 m2 were 1.824 (1.047–3.365), 2.371 (1.109–5.068), and 2.493 (1.193–5.212), respectively]. We found no independent association between eGFR and the risk of CHD. For 4-year all-cause and cardiovascular mortality, integrated discrimination improvement (IDI) was positive when eGFR were added to traditional risk factors (1.51%, P = 0.016, and 1.99%, P = 0.017, respectively). For stroke and CHD events, net reclassification improvements (NRI) were 5.9% (P = 0.012) and 1.8% (P = 0.083) for eGFR, respectively. Conclusions We have established an inversely independent association between eGFR and all-cause mortality, cardiovascular mortality, and stroke in hypertensive patients in rural areas of China. Further, addition of eGFR significantly improved the prediction of 4-year mortality and stroke over and above that of conventional risk factors. We recommend that eGFR be incorporated into prognostic assessment for patients with hypertension in rural areas of China. Limitations We did not have sufficient information on atrial fibrillation to control for the potential covariate. These associations should be further confirmed in future.
Association between plasma inflammatory markers and morphology of coronary artery lesion in patients with coronary artery disease

Xian Wang,Dayi Hu,Shiwei Yang,Jian Zhang,Tan Chen,Shouyan Zhang,

老年心脏病学杂志(英文版) , 2008,
Abstract: Background and Objective The atherosclerotic plaque vulnerability may be related to inflammation, immunity, metabolism and blood clotting. One of the key factors affecting plaque stability is inflammatory reaction. This study was to investigate the relationship between vulnerability of coronary artery plaque evaluated with coronary angiography (CAG), intravascular ultrasound (IVUS) and the levels of plasma inflammatory markers. Methods Fifty-eight consecutive patients with acute coronary syndrome who had coronary lesion of a single vessel were divided into 3 groups based on angiographic morphology of the lesions: type I lesion group (n =16), type II lesion group (n =25) and type III lesion group (n =17). The control group consisted of 17 patients with stable angina. Plasma levels of high sensitivity C reaction protein (hs-CRP), matrix metalloproteinase (MMP, including MMP-2 and MMP-9), CD40 ligand (CD40L) and pregnancy associated plasma protein-A (PAPP-A) were measured by ELISA. Asubgroup of 28 patients (including 18 ACS patients and 10 stable angina control patients) who underwent IVUS study, were analyzed. Results The plasma levels of MMP-2, MMP-9 and PAPP-A in type II lesion group were significantly higher than those in other groups (all P<0.05). In type II lesion group, linear correlation analyses showed significant positive correlation between levels of hs-CRP andMMP-2 (r=0.508); MMP-2 and MMP-9, CD40L, PAPP-A (r=0.647, 0.704 and 0.751, respectively); MMP-9 and CD40L, PAPP-A (r=0.491 and 0.639, respectively); CD40L and PAPP-A (r=0.896). IVUS subgroup analysis showed that the area of plaques and plaque burden in culprit lesion, the incidence of high-risk plaques, remodeling index (RI) and positive remodeling percentage in ACS patients were significantly greater than those in control subgroup (P=0.000, 0.037, 0.028, 0.015 and 0.040, respectively). Compared with control subgroup, the plasma levels of hs-CRP, MMP-2, MMP-9 and PAPP-A were markedly elevated (P=0.033, 0.000, 0.000 and 0.027, respectively). Conclusions CAG and IVUS combined with study on plasma levels of inflammation mediators are helpful in judging the vulnerability of coronary artery plaques.
The Association between Atherosclerotic Renal Artery Stenosis and Acute Kidney Injury in Patients Undergoing Cardiac Surgery
Jingang Yang, Changlin Lu, Li Yan, Xinran Tang, Wei Li, Yuejin Yang, Dayi Hu
PLOS ONE , 2013, DOI: 10.1371/journal.pone.0064104
Abstract: Background Atherosclerotic renal artery stenosis (ARAS) and coronary artery disease (CAD) commonly co-exist. Some patients with unidentified ARAS may undergo cardiac surgery. While acute kidney injury (AKI) is a frequent and serious complication of cardiac surgery, we aim to evaluate the influence of ARAS on the occurrence of postoperative AKI in patients with normal or near-normal baseline renal function following cardiac surgery. Methods A total of 212 consecutive patients undergoing aortography after coronary angiography and cardiac surgery were retrospectively studied for their preoperative and intraoperative conditions. AKI was defined as an absolute increase in serum creatinine of more than or equal to 0.3 mg/dl (≥26.4 μmol/l) or a percentage increase in creatinine of more than or equal to 50% (1.5-fold from baseline) after cardiac surgery. A propensity score-adjusted logistic regression models was used in estimating the effect of ARAS on the risk of postoperative AKI. Results ARAS (≥50%) was observed in 50 (23.6%) patients, and 83 (39.2%) developed AKI after cardiac surgery. A correlation existed between renal artery patency and preoperative–to–postoperative %ΔCr in patients with ARAS (r = 0.297, P<0.0001). The propensity score-adjusted regression model showed the occurrence of postoperative AKI in patients with ARAS was significantly higher than those without ARAS (OR 2.858, 95% CI 1.260–6.480, P = 0.011). Conclusion ARAS is associated with postoperative AKI in patients with normal or near-normal baseline renal function after cardiac surgery.
Ethnic Differences in the Incidence of Hypertension among Rural Chinese Adults: Results from Liaoning Province
Zhaoqing Sun, Liqiang Zheng, Xingang Zhang, Jue Li, Dayi Hu, Yingxian Sun
PLOS ONE , 2014, DOI: 10.1371/journal.pone.0086867
Abstract: Background This study was conducted to examine the differences in the incidence of hypertension and associated risk factors between Mongolian and Han populations in northeast China. Methods A population-based sample of 4753 Mongolian subjects and 20,247 Han subjects aged ≥35 years and free from hypertension at baseline were followed from 2004–2006 to 2010. Incident hypertension was defined as systolic blood pressure≥140 mmHg, diastolic blood pressure ≥90 mmHg, or current use of antihypertensive medication. Results During mean 4.3 years follow-up, a total of 8779 individuals developed hypertension. The age-adjusted incidence of hypertension for Mongolian subjects was 12.64 per 100 person-years, for Han subjects was 9.77 per 100 person-years (P<0.05). The incidence of hypertension was positively correlated with age, physical activity, drinking, body mass index (BMI), family of hypertension and prehypertension in the Han population. In the Mongolian population, hypertension was positively correlated with age, physical activity, education level, drinking, BMI, prehypertension and family history of hypertension. The rates of awareness, treatment and control of hypertension for newly developed cases among both Han and Mongolian populations were low. (36.5% vs. 42.3%, 13.1% vs. 18.2%, 0.7% vs. 1.3%, P<0.05, respectively). Conclusions The incidence rate of hypertension is higher in the Mongolian populations than that in the Han populations, and hypertension in both ethnic populations was associated with similar risk factors. Our results suggest that most newly-diagnosed cases of hypertension are not adequately treated. Improvements in hypertension prevention and control programs in rural China are urgently needed.
From Recurrent Syncope to Sudden Cardiac Death: Clinical Characteristics in a Chinese Patient Carrying a Plakophilin-2 Gene Mutation
Wenling Liu,Xiaoliang Qiu,Wen Liu,Dayi Hu,Tiangang Zhu,Chunling Wang,Dominik Beer,Li Zhang
Case Reports in Cardiology , 2013, DOI: 10.1155/2013/246891
Abstract: We report a case of Arrhythmogenic Right Ventricular Cardiomyopathy (ARVC) which illustrates the natural progression of disease in the absence of availability of an implanted cardiac defibrillator (ICD). Electrocardiograms and cardiac imaging show the progress of ARVC and these clinical milestones of disease are presented herein. 1. Background ARVC is a largely inherited cardiomyopathy that affects mostly young otherwise healthy individuals and is associated with an increased risk of sudden death [1]. The morphological and arrhythmogenic substrate of ARVC predominantly affects the right ventricle (RV) [2] and is characterized by progressive myocardial atrophy with subsequent replacement by fatty and fibrous tissue [3]. Accordingly, the clinical process is also progressive [3, 4]. Here we review the entire progression of a young patient with ARVC from recurrent syncope to sudden cardiac death in the absence of ICD. 2. Case Presentation An 18-year-old Chinese man was sent to the hospital after suffering syncope while playing basketball. After a 12-lead ECG revealed ventricular tachycardia (Figure 1) and a subsequent 24?hr Holter monitor and echocardiography revealed suggestive changes, ARVC was diagnosed according to the 1994 task force criteria [5], and the patient underwent VT ablation. After discharge, episodes of palpitation and syncope persisted during physical activity despite ablation. ICD therapy was strongly recommended to the family who ultimately declined for financial reasons. Bisolol daily and amiodarone were administered with eventual discontinuation of the amiodarone after one year. Episodes of palpitation and syncope disappeared with medical therapy, but the patient suffered sudden cardiac death (SCD) three years later while swimming at 22 years of age. 12-lead ECGs, 24?hr Holter monitoring, and echocardiography were recorded yearly during the 5-year followup. 12-lead ECG showed a markedly fractionated QRS and terminal S wave prolongation. The QRS duration was 130?ms with a large epsilon wave (Figure 2). Holter ECG revealed frequent multifocal PVCs (n3000–4000/24?h) and nonsustained VT with left bundle branch block morphology. His electrocardiogram and echocardiography worsened gradually from the onset of syncopal episodes to death. (Figure 3, Table 1). Holter ECG revealed frequent multifocal PVCs and nonsustained VT (Figure 4). Genetic testing affirmed the diagnosis through the identification of a PKP2 mutation (Figure 5). Table 1: Right ventricle size on echocardiography. Figure 1: Body surface 12-lead ECG documented in 2005. Figure 2:
BFV--BRST Analysis of the Classical and Quantum q-deformations of the sl(2) Algebra
O. F. Dayi
Physics , 1993, DOI: 10.1088/0305-4470/27/1/016
Abstract: BFV--BRST charge for q-deformed algebras is not unique. Different constructions of it in the classical as well as in the quantum phase space for the $q$-deformed algebra sl_q(2) are discussed. Moreover, deformation of the phase space without deforming the generators of sl(2) is considered. $\hbar$-q-deformation of the phase space is shown to yield the Witten's second deformation. To study the BFV--BRST cohomology problem when both the quantum phase space and the group are deformed, a two parameter deformation of sl(2) is proposed, and its BFV-BRST charge is given.
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