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Search Results: 1 - 10 of 329 matches for " Cherian Ajith "
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Ode to Epileptologists!
Cherian Ajith
Annals of Indian Academy of Neurology , 2011,
Electrodiagnostic approach to carpal tunnel syndrome
Cherian Ajith,Kuruvilla Abraham
Annals of Indian Academy of Neurology , 2006,
Abstract: An algorithm for an electrophysiological approach to carpal tunnel syndrome is proposed. This technical note takes into account the standard tests, comparison tests and needle electromyography. If the standard tests are negative, a comparison study can be done to identify cases of minimal or very mild carpal tunnel syndrome (CTS). If comparison studies are negative, CTS can be ruled out and a search for other causes have to be made. If the standard tests are positive, carpal tunnel syndrome can be divided into extreme, severe,moderate and mild cases. Motor comparison study is useful in extreme CTS cases. Needle electromyography is a must in all cases where the standard tests are positive. This streamlined approach allows accurate diagnosis with minimum essential tests.
Muscle channelopathies and electrophysiological approach
Cherian Ajith,Baheti Neeraj,Kuruvilla Abraham
Annals of Indian Academy of Neurology , 2008,
Abstract: Myotonic syndromes and periodic paralyses are rare disorders of skeletal muscle characterized mainly by muscle stiffness or episodic attacks of weakness. Familial forms are caused by mutation in genes coding for skeletal muscle voltage ionic channels. Familial periodic paralysis and nondystrophic myotonias are disorders of skeletal muscle excitability caused by mutations in genes coding for voltage-gated ion channels. These diseases are characterized by episodic failure of motor activity due to muscle weakness (paralysis) or stiffness (myotonia). Clinical studies have identified two forms of periodic paralyses: hypokalemic periodic paralysis (hypoKPP) and hyperkalemic periodic paralysis (hyperKPP), based on changes in serum potassium levels during the attacks, and three distinct forms of myotonias: paramyotonia congenita (PC), potassium-aggravated myotonia (PAM), and myotonia congenita (MC). PC and PAM have been linked to missense mutations in the SCN4A gene, which encodes α subunit of the voltage-gated sodium channel, whereas MC is caused by mutations in the chloride channel gene (CLCN1). Exercise is known to trigger, aggravate, or relieve symptoms. Therefore, exercise can be used as a functional test in electromyography to improve the diagnosis of these muscle disorders. Abnormal changes in the compound muscle action potential can be disclosed using different exercise tests. Five electromyographic (EMG) patterns (I-V) that may be used in clinical practice as guides for molecular diagnosis are discussed.
An unusual cause for weakness in a girl
Khadilkar S,Cherian Ajith,Thomas Sanjeev
Annals of Indian Academy of Neurology , 2007,
Ischemic hyperintensities on T1-weighted magnetic resonance imaging of patients with stroke: New insights from susceptibility weighted imaging
Baheti Neeraj,Cherian Ajith,Chandrasekharan Kesavadas Wattamwar,Thomas Bejoy
Neurology India , 2010,
Abstract: Hyperintensities on T1-weighted magnetic resonance imaging (MRI) in the setting of brain ischemia are usually considered hemorrhagic transformations. Such changes can also be seen due to "incomplete infarction" with selective neuronal loss. Arguments regarding the cause of these T1 hyperintensities have shuttled between gemistocytic astrocyte accumulation, tissue calcification and paramagnetic substance deposition. Susceptibility weighted imaging (SWI), a sensitive modality for detecting paramagnetic agents and blood products, has never been used to resolve this issue. The study was aimed to evaluate the SWI signal changes of T1 hyperintense lesion in stroke patients and understand its usefulness in differentiating a hemorrhagic infarct and an incomplete infarct. All the seven patients with infarct, having hyperintensities on T1 weighted MR imaging seen over the last one year were subjected to SWI. In none of the patients SWI failed to show any blooming. By doing SWI for T1-weighted hyperintensities, we can differentiate hemorrhagic infarct and a non-hemorrhagic "incomplete infarct". This differentiation will immensely help in planning management strategy and prognostication.
Intracerebral hemorrhages in Vogt-Koyanagi-Harada disease
Baheti Neeraj,Cherian Ajith,Kate Mahesh,Krishnan Syam
Neurology India , 2009,
Hemispheric intracranial lipoma with seizure: Look under the carpet
Cherian Ajith,Baheti Neeraj,Menon Ramshekhar,Iyer Rajesh
Neurology India , 2011,
Oxcarbazepine induced worsening of seizures in Jeavons syndrome: Lessons learnt from an interesting presentation
Menon Ramshekhar,Baheti Neeraj,Cherian Ajith,Iyer Rajesh
Neurology India , 2011,
Alpha coma evolving into spindle coma in a case of acute fulminant hepatic failure: What does it signify?
Das Abhijit,Cherian Ajith,Dash G,Radhakrishnan Ashalatha
Annals of Indian Academy of Neurology , 2010,
Abstract: A 44-year-old male developed acute fulminant hepatic failure of unknown etiology and expired within four days. His serial electroencephalograms (EEGs) showed diffuse background slowing on day one, which evolved into "alpha coma" and later into "spindle coma" over the ensuing two days. Such EEG transition is hitherto undescribed in patients with hepatic encephalopathy and gives fresh insight into the etiopathogenesis of specific EEG patterns in diffuse encephalopathy.
An elusive diagnosis: Scedosporium apiospermum infection after near-drowning
Gopinath Malini,Cherian Ajith,Baheti Neeraj,Das Abhijit
Annals of Indian Academy of Neurology , 2010,
Abstract: A 51-year-old male was admitted in our institute following an episode of near-drowning. He later developed ventriculitis and cerebral ring-enhancing lesions. He died following a subarachnoid hemorrhage due to rupture of a mycotic aneurysm involving the right fetal posterior cerebral artery. Scedosporium apiospermum was isolated from the cerebrospinal fluid. Central nervous system invasion by S apiospermum may present insidiously in near-drowning patients and, therefore, requires a high index of suspicion. In cases with the characteristic cerebral ring-enhancing lesions and concomitant ventriculitis, treatment should be instituted while awaiting fungal culture. With this article we intend to alert neurologists, intensivists, and physicians to this near fatal infection, as early identification and prompt treatment with voriconazole may be life saving.
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