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Search Results: 1 - 10 of 5259 matches for " atelectasis and pulmonary edema. "
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Fisiopatología del intercambio gaseoso en el SDRA
Fernández Fernández,R.;
Medicina Intensiva , 2006,
Abstract: ards is produced in a pulmonary edema picture due to increased vascular patency. in this way, the initial alteration consists in an alveolar occupation due to protein rich edema. this occupation reduces the alveolar surface available for gas exchange, increasing the pulmonary areas with poor or null v/q ratio. as ards progresses, vascular phenomena occur that affect the gas exchange differently, giving rise to heterogeneity in the v/q ratio. this situation worsens due to the appearance of areas with null ventilation in relationship with the appearance of atelectasis in lung dependent zones. all these factors form the hypoxemia picture refractory to the increase of the inspired oxygen fraction characteristic of this clinical entity. in this article, we make a review of these physiological mechanisms and the effect on the oxygenation of different ventilatory and drug maneuvers.
Fisiopatología del intercambio gaseoso en el SDRA Pathophysiology of gas exchangein ARDS
R. Fernández Fernández
Medicina Intensiva , 2006,
Abstract: En el síndrome de distrés respiratorio agudo (SDRA) se produce un cuadro de edema pulmonar por aumento de la permeabilidad vascular. De esta forma, la alteración inicial consiste en una ocupación alveolar por edema rico en proteínas, esta ocupación reduce la superficie alveolar disponible para el intercambio gaseoso, incrementando las áreas pulmonares con pobre o nula relación V/Q. A medida que el SDRA progresa, se producen fenómenos vasculares que afectan de forma diferente al intercambio gaseoso, dando lugar a heterogeneidad en la relación V/Q. Esta situación se agrava por la aparición de zonas con nula ventilación en relación con la aparición de atelectasias en zonas dependientes del pulmón. Todos estos factores configuran el cuadro de hipoxemia refractaria al aumento de la fracción de oxígeno inspirado característica de esta entidad clínica. En el presente artículo se hace un repaso de estos mecanismos fisiológicos y del efecto sobre la oxigenación de diferentes maniobras ventilatorias y farmacológicas. ARDS is produced in a pulmonary edema picture due to increased vascular patency. In this way, the initial alteration consists in an alveolar occupation due to protein rich edema. This occupation reduces the alveolar surface available for gas exchange, increasing the pulmonary areas with poor or null V/Q ratio. As ARDS progresses, vascular phenomena occur that affect the gas exchange differently, giving rise to heterogeneity in the V/Q ratio. This situation worsens due to the appearance of areas with null ventilation in relationship with the appearance of atelectasis in lung dependent zones. All these factors form the hypoxemia picture refractory to the increase of the inspired oxygen fraction characteristic of this clinical entity. In this article, we make a review of these physiological mechanisms and the effect on the oxygenation of different ventilatory and drug maneuvers.
Pulmonary artery aneurysm mimicking a lung mass
Shameem M,Bhargava R,Ahmad Z,Shah N
Lung India , 2007,
Abstract: We are describing a case of idiopathic pulmonary artery aneurysm, which pre-sented to us with complaints of shortness of breath and chest pain. Chest -X ray showed a homogenous opacity occupying left mid and lower zone, CECT thorax revealed a pulmonary artery aneurysm which was compressing the left bronchus and causing atlectasis. The patient was managed surgically
Negative Pressure Pulmonary Edema on Extubation of a 9-Month-Old Baby Boy  [PDF]
Dennis E. Feierman, Katerina Svigos, Evan P. Salant, Agnes Miller
Open Journal of Anesthesiology (OJAnes) , 2015, DOI: 10.4236/ojanes.2015.55018
Abstract: Background: Negative Pressure Pulmonary Edema (NPPE) is an uncommon, but well recognized clinical entity that continues to be reported as a complication of upper airway obstructions during induction or emergence. It results from the negative intrathoracic pressure generated with spontaneous ventilation with concurrent upper airway obstruction. Aim: To present an unusual case of NPPE and review the pathophysiology and treatment. Case: It usually occurs in young healthy athletic adults. We are reporting NPPE in a nine-month-old ex-premature baby. We discuss his intraoperative events leading to NPPE, subsequent intraoperative course and treatment. Conclusion: NPPE needs to be promptly recognized and treated. If the edema resolves, the patient can be successfully extubated, but should be observed overnight.
Independent Lung Ventilation for Reexpansion Pulmonary Edema*  [PDF]
Sheetal Garg, Harinder Singh Bedi, Melchisedec Singh, Valsa Abraham
World Journal of Cardiovascular Surgery (WJCS) , 2013, DOI: 10.4236/wjcs.2013.35033
Abstract: Reexpansion pulmonary oedema (RPE) is a rare but potentially fatal complication with no clear cut guidelines for its management. When the injury to the lung is primarily one sided, conventional modes of ventilation can be ineffective and at times harmful. Selective or independent lung ventilation (ILV) is one of the therapeutic modality that can be used for the treatment of such cases. We report the successful treatment of reexpansion pulmonary oedema in 19-year-old boy using independent lung ventilation.
Preoperative Atrial Fibrillation and T-Wave Inversion Associated with Postoperative Pulmonary Edema  [PDF]
Thomas Hong, Benson George, Mohamad Hashim, Dennis E. Feierman
Open Journal of Anesthesiology (OJAnes) , 2016, DOI: 10.4236/ojanes.2016.610027
Abstract:
Background: Atrial fibrillation (AF) and T wave inversion (TWI) are occasionally found on pre-operative electrocardiograms (EKG) of patients with long standing hy-pertension (HTN) associated with left ventricular hypertrophy (LVH). Each of these two pathologies, AF and LVH, can impose negative hemodynamic effects on the car-diac function. Case: We present a case of a patient with AF, TWI and long standing HTN that went into post-operative pulmonary edema. Conclusion: Even returning to base line poorly controlled hypertension, and even more so during a hypertensive cri-sis, their concurrent presence may inflict even more detrimental effect manifested by elevation of pulmonary venous pressure and pulmonary edema.
Re-expansion pulmonary edema in a boy with spontaneous pneumothorax during an influenza B virus infection  [PDF]
Toru Watanabe
Open Journal of Pediatrics (OJPed) , 2012, DOI: 10.4236/ojped.2012.24042
Abstract: Re-expansion Pulmonary Edema (RPE) is an uncommon complication following rapid re-expansion of the lungs, and the most common disorder associated with RPE is spontaneous pneumothorax. The majority of patients with RPE associated with pneumothorax have been adults; only 4 cases have been reported in children. We present a patient who developed RPE after treatment of spontaneous pneumothorax that occurred during an influenza B virus infection. His condition improved only with oxygen supplementation and intravenous corticosteroids. Infection with influenza B virus leads to increased production of lung epithelial oxidants, which might have contributed to the development of RPE in our patient.
Edema pulmonar por presión negativa: a propósito de 3 casos
Ortiz-Gómez,J. R.; Plaja-Marti,I.; Sos-Ortigosa,F.; Pérez-Cajaraville,J. J.; Arteche-Andrés,M. A.; Bengoechea,C.; Lobo-Palanco,J.; Ahmad-Al-Ghool,M.;
Anales del Sistema Sanitario de Navarra , 2006, DOI: 10.4321/S1137-66272006000300010
Abstract: negative pressure pulmonary edema is a complication, described since 1977, caused by upper airway obstruction in both children and adults. although its aetiopathogeny is multifactorial, especially outstanding is excessive negative intrathoracic pressure caused by the forced spontaneous inspiration of a patient against a closed glottis, that causes high arteriole and capillary fluid pressures that favor transudation into the alveolar space the resulting pulmonary edema can appear a few minutes after the obstruction of the airway or in a deferred way after several hours. the clinical manifestations are potentially serious, but normally respond well to treatment with supplemental oxygen, positive pressure mechanical ventilation and diuretics. diagnostic suspicion is important for acting promptly. we report three clinical cases with acute negative pressure pulmonary edema.
Edema pulmonar agudo como primera manifestación de hipertiroidismo en una mujer embarazada
Díaz N,Rienzi; Silva G,Danilo;
Revista médica de Chile , 2008, DOI: 10.4067/S0034-98872008000300012
Abstract: we report a 36 year-old pregnant woman who presented with acute pulmonary edema in the absence of preexisting cardiac disease. on admission she was on sinus rhythm and her blood pressure was mildly elevated. no cardiac abnormalities were detected by color doppler echo car diography and no ischemic changes were seen on the electrocardiogram. cardiac enzymes were normal. thyroid function tests were diagnostic for hyperthyroidism. she was treated with propylthiouracil and propranolol and discharged in good conditions seven days after admission. this case emphasizes the need to consider hyperthyroidism as the cause of unexplained pulmonary edema in young patients with no history of heart disease who present with heart failure
Reexpansion pulmonary edema- A case report
Janmeja A,Mohapatra P,Saini M,Khurana A
Lung India , 2007,
Abstract: A middle aged male presented to us with right side pneumothorax. Two hours after insertion of intercostals tube he felt discomfort and increased breathlessness. His chest examination showed crepitations. Chest radiograph showed partial expansion of right lung with opacity in right lower zone consistent with unilateral pulmonary edema. He was managed conservatively and improved with complete resolution on chest radiograph. Reexpansion pulmonary edema is an uncommon complication of the treatment of lung atelectasis, pleural effusion or pneumothorax and patho-genesis is unclear.
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