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Search Results: 1 - 10 of 376638 matches for " James P. de;Novis "
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Síndrome 'one-and-a-half': considera??es anatomoclínicas a propósito de um caso
André, Charles;Castro, Ana Lúcia Z. de;Vincent, Maurice B.;Mattos, James Pitágoras de;Maranh?o Filho, Péricles de A.;Novis, Sérgio A. Pereira;
Arquivos de Neuro-Psiquiatria , 1989, DOI: 10.1590/S0004-282X1989000300020
Abstract: a 36 years_old man was admitted in september 1987. for 5 years he suffered from 4 recurrent episodes of throbbing headache, tinnitus, nausea, diplopia and divergent strabismus to which a facial palsy was recently added. in all episodes, the symptoms disappeared spontaneously and completely. a neuro-ophthalmological examination at admission disclosed an exotropia of the right eye, gaze paralysis to the left, paralysis of adduction of the left eye and preserved right eye abduction which triggered a rhythmic horizontal nystagmus. the upward and downward gazes and the convergence were well preserved. moreover, there was a left peripheral facial palsy, and babinski sign at the right side. auditory evoked potenciais were slowed at the mesencephalopontine transition. ct scan showed a low-density area with no contrast enhancement at the left pontine tegmentum and a left anterolateral atrophy of the pons. csf exmination showed increase in protein content and increase in the igg content. additional investigation included a dopplerometry of the cervical arteries, a panangiography and a bidimensional echocardiography which were normal. diagnosis of one-and-a-half syndrome was made, possibly secondary to multiple sclerosis, and immunos-supressive therapy was initiated.
Movement disorders in 28 HIV-infected patients
Mattos, James Pitágoras de;Rosso, Ana Lúcia Zuma de;Corrêa, Rosalie Branco;Novis, Sérgio A.P.;
Arquivos de Neuro-Psiquiatria , 2002, DOI: 10.1590/S0004-282X2002000400002
Abstract: from 1986 to 1999, 2460 hiv-positive inpatients were seen in our hospital. neurological abnormalities were detected in 1053 (42.8%) patients. in this group, 28 (2.7%) had involuntary movements, 14 (50%) with secondary parkinsonism, six (21.4%) with hemichorea/hemiballismus, four (14.2%) with myoclonus, two (7.2%) with painful legs and moving toes, one (3.6%) with hemidystonia and one (3.6%) with holmes' tremor. the hiv itself (12 patients), toxoplasmosis of the midbrain (1) and metoclopramide-related symptoms (1) were the most probable causes for the parkinsonism. all patients with hemichorea/hemiballismus were men and in all of them toxoplasmosis of the basal ganglia, mostly on the right side, was the cause of the involuntary movements. generalized myoclonus was seen in two patients and they were due to toxoplasmosis and hiv-encephalopathy respectively; two others presented with spinal myoclonus. the two patients with painful legs and moving toes had an axonal neuropathy. the patient with hemidystonia suffered from toxoplasmosis in the basal ganglia and the patient with holmes' tremor had co-infection with tuberculosis and toxoplasmosis affecting the midbrain and cerebellum. we conclude that hiv-infected patients can present almost any movement disorder. they can be related to opportunistic infections, medications, mass lesions and possibly to a direct or indirect effect of the hiv itself.
Movement disorders in 28 HIV-infected patients
Mattos James Pitágoras de,Rosso Ana Lúcia Zuma de,Corrêa Rosalie Branco,Novis Sérgio A.P.
Arquivos de Neuro-Psiquiatria , 2002,
Abstract: From 1986 to 1999, 2460 HIV-positive inpatients were seen in our Hospital. Neurological abnormalities were detected in 1053 (42.8%) patients. In this group, 28 (2.7%) had involuntary movements, 14 (50%) with secondary parkinsonism, six (21.4%) with hemichorea/hemiballismus, four (14.2%) with myoclonus, two (7.2%) with painful legs and moving toes, one (3.6%) with hemidystonia and one (3.6%) with Holmes' tremor. The HIV itself (12 patients), toxoplasmosis of the midbrain (1) and metoclopramide-related symptoms (1) were the most probable causes for the parkinsonism. All patients with hemichorea/hemiballismus were men and in all of them toxoplasmosis of the basal ganglia, mostly on the right side, was the cause of the involuntary movements. Generalized myoclonus was seen in two patients and they were due to toxoplasmosis and HIV-encephalopathy respectively; two others presented with spinal myoclonus. The two patients with painful legs and moving toes had an axonal neuropathy. The patient with hemidystonia suffered from toxoplasmosis in the basal ganglia and the patient with Holmes' tremor had co-infection with tuberculosis and toxoplasmosis affecting the midbrain and cerebellum. We conclude that HIV-infected patients can present almost any movement disorder. They can be related to opportunistic infections, medications, mass lesions and possibly to a direct or indirect effect of the HIV itself.
Apresenta??o neurológica incomum no curso de paracoccidioidomicose relato de um caso
Corrêa, Rosalie Branco;Puccioni-Sohler, Marzia;Artemenko, Sofia R. T.;Nogueira, Susie A.;Mattos, James Pitágoras de;Novis, Sergio A. P.;
Arquivos de Neuro-Psiquiatria , 1991, DOI: 10.1590/S0004-282X1991000400016
Abstract: a case of paracoccidioidomycoses of the central nervous system and movement disorders is reported in this paper, and neurological aspects are discussed.
Paroxysmal dystonia and neuromyelitis optica
Schmidt, Felipe R.;Costa, Flavio Henrique R.;Silva, Fernanda M.L.C.;Maultasch, Henryk;Rosso, Ana Lucia;Nicaretta, Denise H.;Mattos, James P. de;Novis, Sergio A.P.;Alves-Leon, Soniza V;
Arquivos de Neuro-Psiquiatria , 2012, DOI: 10.1590/S0004-282X2012005000011
Abstract: paroxysmal dyskinesias (pd) are thought to be rare movement disorders. the overwhelming majority of reported cases are primary. secondary pd has seen reported to occur in some conditions, mainly in multiple sclerosis and head trauma. the anatomic origin of the lesion is also rarely seen at the spinal cord. our objective was to describe four patients with paroxysmal dystonia secondary to spinal lesions during the recovering phase of a neuromyelitis optica (nmo) bout. in the reviewed literature, we do not find any report of pd related to nmo.
Distonias: aspectos clínicos e terapêuticos em 64 pacientes
Mattos, James Pitágoras de;Rosso, Ana Lúcia Zuma de;Novis, Sérgio;
Arquivos de Neuro-Psiquiatria , 1996, DOI: 10.1590/S0004-282X1996000100005
Abstract: the experience with 64 patients with dystonia seen at the extrapyramidal diseases sector of the neurology department of the hospital universitário clementino fraga filho of the ufrj is presented as well as the pertinent review of the literature. the five-and-a-half-year of follow-up showed that 33 were male and 31 female; 48 were white, 10 mulatto and 6 negro; the mean time of disease was 9 years and 8 months. according to the distribution of the movement disorder, 30 (46.9%) were focal, 17 (26.6%) segmental, 13 (20.3%) generalized, 3 (4.7%) hemidystonia and 1 (1.5%) multifocal. in 11 (17.2%) the age of onset was before 12 years old, in 6 (9.4%) between 13 and 20 years old, and in 47 (73.4%) after 20 years old. according to the etiology, 39 (60.9%) were idiopathic sporadic, 6 (9.4%) were idiopathic familial and 19 (29.7%) were symptomatic. the therapeutical approach used in these patients were anticholinergic drugs, dopaminergic agonists or antagonists and baclofen, alone or associated with anticholinergic drugs for generalized dystonia. the authors conclude that botulinum toxin type a is the most valuable therapeutic agent in the treatment of focal dystonia.
Toxina botulínica no blefaroespasmo, no espasmo hemifacial e na distonia cervical: resultados em 33 pacientes
Novis, Sérgio Ap.;Mattos, James Pitágoras de;Rosso, Ana Lúcia Zuma de;
Arquivos de Neuro-Psiquiatria , 1995, DOI: 10.1590/S0004-282X1995000300006
Abstract: the effects of botulinum toxin type a were studied in 33 patients with dystonia (12 blepharospasms, 10 hemifacial spasms and 11 spasmodic torticollis). a rate scale was used to evaluate the severity of the dystonic movements, before and two weeks after each injection. among blepharospasm patients, eight were female and four were male; the mean age was 57.7 years; the mean time of the disease duration was four years. three had familial history for similar disease; nine were essential and three had used neuroleptic drugs (tardive dystonia). the mean dose used was 51.3 u, with a mean time of benefical effects of 2.8 months. for 22 injections and reinjections, 14 (63.7%) showed an excellent result, five (22.7%) good and three (13.6%) null. in the hemifacial spasm group, eight were female and two male; the mean age was 52.6 years; the mean time of the disease duration was 7.4 years; eight were essential and two post-paralytic. the mean dose used was 32 u. from the total of 15 injections and reinjections, all of them (100%) had an excellent result, with a mean time of benefical effect of 3.4 months. among the cervical dystonic patients, eight were male and three female; the mean age was 44.2 years; the mean time of the disease duration was 12.2 years; six had essential dystonia, three had used neuroleptic drugs and two had familial history for similar disease. the mean dose used was 238.6 u, with the mean duration of effect of 3.5 months. from the total of 20 injections and reinjections, 18 (90%) had good result, one (5%) mild and one (5%) null. ptosis, facial palsy and dysphagia were the most common side effects seen. we conclude that botulinum toxin is effective for the treatment of such disorders.
Spinal myoclonus: report of four cases
Mattos, James Pitágoras de;Rosso, Ana Lúcia Z.;Carneiro, Antonio J. V.;Novis, Sergio;
Arquivos de Neuro-Psiquiatria , 1993, DOI: 10.1590/S0004-282X1993000400012
Abstract: four cases of spinal myoclonus are described, three males and one female. the mean age was 51 years (28-75 years). the mean time between the onset of the myelopathy and the myoclonic jerks was 4.3 months (1-8 months). the involuntary movements were determined by trauma, devic's disease, tuberculous myelopathy and tumor. three patients had spastic paraplegia with bilateral myoclonus more evident on the right side. the fourth patient had a flaccid paraplegia with symmetrical jerks. the data suggest that different processes (trauma, demyelinating, infection and tumor) affecting the spinal cord may cause the same type of involuntary movements.
Involuntary movements and AIDS: report op seven cases and review of the literature
Mattos, James Pitágoras de;Rosso, Ana Lúcia Z.;Corrêa, Rosalie Branco;Novis, Sérgio;
Arquivos de Neuro-Psiquiatria , 1993, DOI: 10.1590/S0004-282X1993000400011
Abstract: we studied 1086 aids patients in the last six years. of these 389 (35.82%) had neurological manifestation and 7 (1.8%) male patients had abnormal involuntary movements (parkinsonism in 3, hemichorea-hemiballism in 2, spinal myoclonus in 1 and rubral tremor in another). all patients were men, 5 white and 2 black. four were homosexual, 2 drug-users and 1 bisexual. the mean age was 33.14 years. the time between aids diagnosis and the onset of movement disorders was 23.8 months in 5 patients and in 2 it was the first symptom. the parkinsonian patients did not show any opportunistic infection in conection with the neurological symptoms but in the remaining four cases this relationship was suggested. the data showed that not only the opportunistic infection but also the aids virus may play an important role on the development of involuntary movements.
Segmental myoclonus and basilar artery giant aneurysm case report
Mattos, J. Pitágoras de;Rosso, Ana L. Zuma de;Zayen, Eduardo;Novis, Sérgio A. P.;
Arquivos de Neuro-Psiquiatria , 1992, DOI: 10.1590/S0004-282X1992000400019
Abstract: a 70 years-old man was admitted at our hospital because of unstable angina pectoris. he had essential hypertension and right hemiplegia from a ischemic stroke two years before admission. on neurologic examination, it was found mental disorientation, unstable emotionality, right spastic hemiparesis with right babinski sign, and segmental myoclonus affecting the superior lip and the palate (palatal nystagmus) on the right side. on the ct scan, a giant aneurysm of the basilar artery was detected. we conclude that the segmental myoclonus could be explained by ischemic lesions in the guillain - mollaret triangle.
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