Publish in OALib Journal

ISSN: 2333-9721

APC: Only $99


Any time

2019 ( 28 )

2018 ( 187 )

2017 ( 206 )

2016 ( 262 )

Custom range...

Search Results: 1 - 10 of 144439 matches for " Fermin F. Chaina-Chura "
All listed articles are free for downloading (OA Articles)
Page 1 /144439
Display every page Item
Factors Related to the Level of Perceived Quality of Academic and Administrative Services by Users: Case of the National University of the Altiplano, Puno, Peru  [PDF]
Juan Inquilla-Mamani, Fermin F. Chaina-Chura
Open Access Library Journal (OALib Journal) , 2019, DOI: 10.4236/oalib.1105467
The purpose of this research is to determine the conditioning factors of quality of academic and administrative services perceived by users of the National University of the Altiplano (UNA-Puno). In the methodological part, the different statistics are presented as Kendall’s Taub test, Factorial analysis of the components, Factors extraction, Varimax rotation, T test and correlation estimates and multiple linear regression. The study is of transversal design the study sample is made up of students, teachers and administrative staff of the UNA-P, of a simple random type. The measurement instrument applied was the Servqual Questionnaire. For the processing and statistical analysis of the information, the SPSS v. 22 program. The results obtained through the reliability analysis of the Servqual model show us a Cronbach alpha of 0.709, which means that both the instrument and its attributes are reliable and consistent to measure the quality of the service. Considered in the model determine in 67.11% the levels of service quality, R2 is 0.746 and value of F = 0.000. Therefore, the factors identified in the analysis directly and significantly affect the level of perceived quality.
Beach Closures in Oakland County, Michigan: Using GIS as an Investigative Tool
Jeanine Chura McCloskey
Solstice : Electronic Journal of Geography and Mathematics , 2002,
Abstract: The link to the fulltext on a current website is given below. Should that link fail to work, please go to this persistent URL and download the associated file for this issue. http://deepblue.lib.umich.edu/handle/2027.42/58219While you are there, take a look around and see if there are other issues and documents that are related to your interests!
Stacking a 4D geometry into an Einstein-Gauss-Bonnet bulk
Carlos Barcelo,Roy Maartens,Carlos F. Sopuerta,Fermin Viniegra
Physics , 2002, DOI: 10.1103/PhysRevD.67.064023
Abstract: In Einstein gravity there is a simple procedure to build D-dimensional spacetimes starting from (D-1)-dimensional ones, by stacking any (D-1)-dimensional Ricci-flat metric into the extra-dimension. We analyze this procedure in the context of Einstein-Gauss-Bonnet gravity, and find that it can only be applied to metrics with a constant Krestschmann scalar. For instance, we show that solutions of the black-string type are not allowed in this framework.
(0,2) String Compactifications: The Higgs Mechanism
Fermin Aldabe
Physics , 1995,
Abstract: We present the Higgs mechanism in (0,2) compactifications. The existence of a vector bundle data duality (VBDD) in $(0,2)$ compactifications which is present at the Landau-Ginzburg point allows us to connect in a smooth manner theories with different gauge groups with the same base manifold and same number of effective generations. As we move along the Kahler moduli space of the theories with $E_6$ gauge group, some of the gauginos pick up masses and break the gauge group to $SO(10)$ or $SU(5)$.
Quantum Solitons Lead to Yukawa Coupling
Fermin Aldabe
Physics , 1995,
Abstract: Recently, it was shown that zero modes in semiclassical soliton models do not lead to Yukawa couplings. We show that taking into account the contributions of the quantum soliton into the renormalization scheme, which cannot be done in semiclassical treatments, leads to a Yukawa coupling. A similar analysis should be possible for the Skyrmion, renewing the hope, that this model will lead to a correct description of hadron physics.
Can Plane Wave Modes be Physical Modes in Soliton Models?
Fermin Aldabe
Physics , 1995, DOI: 10.1016/0370-2693(95)00385-X
Abstract: I show that plane waves may not be used as asymptotic states in soliton models because they describe unphysical states. When asymptotic states are taken to be physical there is no T-matrix of $\cO(1)$.
Brownian Motion of Solitons in the $Φ^4$ Model
Fermin Aldabe
Physics , 1995, DOI: 10.1142/S0217979296000519
Abstract: We derive an expression for the correlation function of the random force on a soliton which is consistent with the constraints needed to integrate out the zero modes which appear due to the broken translational symmetry of the soliton solution. It is shown that when the constraint does not commute with the operator which defines the correlation function, i.e. when the operator is not physical, only low frequency phonons contributions may be considered. On the contrary, when the correlation function of the random force on the soliton is constructed with physical operators one may also include in a correct manner the contributions from the optical phonons.
Collective Coordinates and the Absence of Yukawa Coupling in the Classical Skyrme Model
Fermin Aldabe
Physics , 1996, DOI: 10.1016/S0370-2693(96)01235-X
Abstract: In systems with constraints, physical states must be annihilated by the constraints. We make use of this rule to construct physical asymptotic states in the Skyrme model. The standard derivation of the Born terms with asymptotic physical states shows that there is no Yukawa coupling for the Skyrmion. We propose a remedy tested in other solitonic models: A Wilsonian action obtained after integrating the energetic mesons and where the Skyrmion is a quantum state should have a Yukawa coupling.
Serial analysis of gene expression reveals differential expression between endometriosis and normal endometrium. Possible roles for AXL and SHC1 in the pathogenesis of endometriosis
Hiroshi Honda, Fermin F Barrueto, Jean Gogusev, Dwight D Im, Patrice J Morin
Reproductive Biology and Endocrinology , 2008, DOI: 10.1186/1477-7827-6-59
Abstract: In order to better understand the pathogenesis of endometriosis, we have used Serial Analysis of Gene Expression (SAGE) to identify genes differentially in this disease by studying three endometriotic tissues and a normal endometrium sample. Promising candidates (AXL, SHC1, ACTN4, PI3KCA, p-AKT, p-mTOR, and p-ERK) were independently validated by immunohistochemistry in additional normal and endometriotic tissues.We identified several genes differentially expressed between endometriosis and normal endometrium. IGF2, ACTN4, AXL, and SHC1 were among the most upregulated genes. Comparison of the endometriosis gene expression profiles with the gene expression patterns observed in normal human tissues allowed the identification of endometriosis-specific genes, which included several members of the MMP family (MMP1,2,3,10,11,14). Immunohistochemical analysis of several candidates confirmed the SAGE findings, and suggested the involvement of the PI3K-Akt and MAPK signaling pathways in endometriosis.In human endometriosis, the PI3K-Akt and MAPK signaling pathways may be activated via overexpression of AXL and SHC1, respectively. These genes, as well as others identified as differentially expressed in this study, may be useful for the development of novel strategies for the detection and/or therapy of endometriosis.Endometriosis is a common gynecological disorder that affects 5–10% of women of reproductive age and that causes dysmenorrhea, chronic pelvic pain, and infertility [1]. Despite numerous studies on endometriosis, its etiology and pathogenesis have not yet been fully elucidated. The disease is characterized by the presence of endometrial epithelial and stromal cells outside the uterine cavity. Biologically, these ectopic cells are quite distinct from their eutopic counterparts. Different responses to steroid hormones, growth factors and cytokines, chromosomal aberrations [2-4], Loss of heterozygosity (LOH), or allelic loss of specific chromosomal regions [5-8] have b
Down-regulation of cell surface CXCR4 by HIV-1
Bongkun Choi, Paul J Gatti, Cesar D Fermin, Sandor Vigh, Allyson M Haislip, Robert F Garry
Virology Journal , 2008, DOI: 10.1186/1743-422x-5-6
Abstract: Infection of human T-lymphoblastoid cell line RH9 with HIV-1 resulted in down-regulation of cell surface CXCR4 expression. Down-regulation of surface CXCR4 correlated temporally with the increase in HIV-1 protein expression. CXCR4 was concentrated in intracellular compartments in H9 cells after HIV-1 infection. Immunofluorescence microscopy studies showed that CXCR4 and HIV-1 glycoproteins were co-localized in HIV infected cells. Inducible expression of HIV-1 envelope glycoproteins also resulted in down-regulation of CXCR4 from the cell surface.These results indicated that cell surface CXCR4 was reduced in HIV-1 infected cells, whereas expression of another membrane antigen, CD3, was unaffected. CXCR4 down-regulation may be due to intracellular sequestering of HIV glycoprotein/CXCR4 complexes.Chemokine receptors are seven-transmembrane G-protein-coupled receptors that upon ligand binding transmit signals, such as calcium flux, resulting in chemotactic responses [1-3]. Chemokine receptors are divided into four families that reflect differential binding of the CXC, CC, CX3C and XC subfamilies of chemokines [4]. Several members of the chemokine receptor family function as coreceptors with the primary receptor CD4 to allow entry of various strains of human immunodeficiency virus type 1 (HIV-1) into the cells [5-8]. T-cell-tropic X4 HIV-1 use CD4 and chemokine receptor CXCR4 for entry into target cells, whereas macrophage-tropic R5 HIV-1 use CD4 and chemokine receptor CCR5. Dual-tropic strains can use either CCR5 and CXCR4 as co-receptors. In addition, CCR3, CCR2, CXCR6 (Bonzo/STLR6) among other chemokine receptors can function as coreceptors and support infection by a more restricted subset of macrophage-tropic or dual-tropic HIV strains [9,5,12].CXCL12 (stromal derived factor 1 α/β, SDF-1α/β) is the natural ligand for CXCR4, whereas CC chemokines, CCL3 (macrophage inflammatory factor 1α, MIP-1α/chemokine LD78α), CCL3-L1 (LD78β), CCL4 (MIP-1β), and CCL5 (RANTES), are li
Page 1 /144439
Display every page Item

Copyright © 2008-2017 Open Access Library. All rights reserved.