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Search Results: 1 - 10 of 7663 matches for " Claude Marcus "
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Eating behaviour patterns in Chinese children aged 12-18 months and association with relative weight - factorial validation of the Children's Eating Behaviour Questionnaire
Ying-Ting Cao, Viktoria Svensson, Claude Marcus, Jing Zhang, Jian-Duan Zhang, Tanja Sobko
International Journal of Behavioral Nutrition and Physical Activity , 2012, DOI: 10.1186/1479-5868-9-5
Abstract: 219 questionnaires were filled out by the caregivers, approached in community health care centers in two cities in China. BMI of each child was calculated and converted to BMI SDS. Factor validation (Principal Component Analysis, exploratory factor analysis) on all CEBQ items was performed and gender difference in eating behaviours was examined. Correlations between eating behaviours and the child's BMI SDS were analyzed by linear regression analysis controlling for gender, parental combined weight, and education.The factor analysis revealed a seven-factor solution, with factor 'food responsiveness' (FR) split into two. 'Satiety responsiveness' (SR) and 'Enjoyment of food' (EF) factors were not detected. Interestingly, boys scored higher than girls in the FR scales, whereas girls had a higher score in 'food fussiness' (FF) scale.We conclude that although a valuable psychometric instrument, CEBQ might be affected by age and cultural differences. Therefore, adjusting it in order to fit the Chinese population was suggested. We did not find an association between eating behaviours and children's BMI SDS, when it was controlled for gender and parental weight.Childhood obesity has become one of the most serious public health challenges worldwide in the last century, with 42 million overweight children under the age of five. The vast majority (80%) of these live in low and middle-income countries [1]. This trend has affected China and the prevalence of childhood obesity in some urban Chinese populations is already reached similar levels of the developed countries [2]. In 2005, total 7.73% of Chinese youth were overweight and 3.71% of them were obese. Partly explained by the explosive economic development and improvement of living conditions in China [3], general raise in income in China today enables a decreasing consumption of cereals and starchy roots, and an increasing consumption of high-caloric foods [4]. In addition, fast food and snack consumption together with incr
Obesity related eating behaviour patterns in Swedish preschool children and association with age, gender, relative weight and parental weight - factorial validation of the Children's Eating Behaviour Questionnaire
Viktoria Svensson, Linda Lundborg, Yingting Cao, Paulina Nowicka, Claude Marcus, Tanja Sobko
International Journal of Behavioral Nutrition and Physical Activity , 2011, DOI: 10.1186/1479-5868-8-134
Abstract: Parents to 174 children aged 1-6 years (50% girls, mean age 3.8 years), recruited from five kindergartens in Stockholm, completed the Swedish version of the CEBQ. Data on children's weight and height, parental weight, height and educational level was collected. Children's relative weight was calculated for a subpopulation (mean BMI SDS -0.4, n = 47). Factorial validation (Principal Component Analysis) on all CEBQ items was performed. Differences in eating behaviours by age, gender and parental weight were examined. Correlations between eating behaviours and the child's BMI SDS were analysed controlling for age, gender, parental weight and education in linear regression analyses.The factor analysis revealed a seven factor solution with good psychometric properties, similar to the original structure. The behaviour scales 'overeating'/'food responsiveness', 'enjoyment of food' and 'emotional undereating' decreased with age and 'food fussiness' increased with age. Eating behaviours did not differ between girls and boys. The children's relative weight was not related to any of the eating behaviours when controlling for age, gender, parental weight and education, and only associated with parental weight status.Our results support the use of the CEBQ as a psychometric instrument for assessing children's eating behaviours in Swedish children aged 1-6 years. Measuring obesity related eating behaviours in longitudinal and interventional studies would offer opportunities for studying causal effects of eating behaviours in the development of obesity in children.One of the strongest risk factors for childhood obesity is parental overweight and obesity [1,2]. To prevent a further increase of obesity in children, there is an urgent need for evidence-based interventions, targeting families in different risk groups [3,4]. Previous studies have suggested that weight problems in children can, to some extent, be explained by individual differences in eating style [5,6]. It would be imp
Generating Net Forces from Backgrounds of Randomly Created Waves  [PDF]
Claude Gauthier
Journal of Modern Physics (JMP) , 2014, DOI: 10.4236/jmp.2014.516158
Abstract: We examine the possibility of generating net forces on concave isolated objects from backgrounds consisting of randomly created waves carrying momentum. This issue is examined first for waves at the surface of a liquid, and second for quantum vacuum electromagnetic waves, both in relation with a one-side-open rectangular structure whose interior embodies a large number of parallel reflecting plates. Using known results about the Casimir-like effect and the original Casimir effect for parallel plates, we explain why and how such rectangular hollow structures should feel net oriented forces. We briefly describe real systems that would allow testing these theoretical results.
Wave-Particle Duality in Einstein-de Broglie Programs  [PDF]
Claude Elbaz
Journal of Modern Physics (JMP) , 2014, DOI: 10.4236/jmp.2014.518213
Abstract: The standard model of particle physics forms a consistent system for universe description. After following quantum mechanics, it derives particles from relativistic quantum fields. Since it does not include gravitation, it describes only one aspect of the universe. In extension of general relativity, Einstein had proposed a symmetrical and complementary approach of physics. In his program, he privileged a relativist field based on representations for physical phenomena, before a precise mathematical description. It allows completing and unifying the universe description, like both eyes for relief vision, and both ears for stereophonic audition. We propose to show it with many simple examples.
Gravitation and Electromagnetism Conciliated Following Einstein’s Program  [PDF]
Claude Elbaz
Journal of Modern Physics (JMP) , 2015, DOI: 10.4236/jmp.2015.65072
Abstract: The Einstein’s program permits to conciliate gravitation and electromagnetism. Besides the standard model, it forms a consistent system for universe description, founded upon a scalar field propagating at the speed of light c. Matter corresponds to standing waves. Adiabatic variations of frequencies lead to electromagnetic interaction constituted by progressive waves. Classical domain corresponds to geometrical optics approximation, when frequencies are infinitely high, and then hidden. As interactions for matter, Gravitation and Electromagnetism derive from variations of its energy E = mc2. Electromagnetic interaction energy derives from mass variation dE = c2dm, and gravitation from speed of light variation dE = mdc2. Contrarily to gravitation, only electromagnetic interaction serves as a bridge between classical and quantum frames, since it leans directly upon the wave property of matter: its energy dE = hdν = c2dm derives from variations of matter energy E = hν = mc2.
Genetic Variance in the Adiponutrin Gene Family and Childhood Obesity
Lovisa E. Johansson, Lina M. Johansson, Pernilla Danielsson, Svante Norgren, Stina Johansson, Claude Marcus, Martin Ridderstr?le
PLOS ONE , 2009, DOI: 10.1371/journal.pone.0005327
Abstract: Aim The adiponutrin gene family consists of five genes (PNPLA1-5) coding for proteins with both lipolytic and lipogenic properties. PNPLA3 has previously been associated with adult obesity. Here we investigated the possible association between genetic variants in these genes and childhood and adolescent obesity. Methods/Results Polymorphisms in the five genes of the adiponutrin gene family were selected and genotyped using the Sequenom platform in a childhood and adolescent obesity case-control study. Six variants in PNPLA1 showed association with obesity (rs9380559, rs12212459, rs1467912, rs4713951, rs10947600, and rs12199580, p<0.05 after adjustment for age and gender). Three variants in PNPLA3 showed association with obesity before, but not after, adjustment for age and gender (rs139051, rs12483959, and rs2072907, p>0.05). When analyzing these SNPs in relation to phenotypes, two SNPs in the PNPLA3 gene showed association with insulin sensitivity (rs12483959: β = ?0.053, p = 0.016, and rs2072907: β = ?0.049, p = 0.024). No associations were seen for PNPLA2, PNPLA4, and PNPLA5. Conclusions Genetic variation in the adiponutrin gene family does not seem to contribute strongly to obesity in children and adolescents. PNPLA1 exhibited a modest effect on obesity and PNPLA3 on insulin sensitivity. These data, however, require confirmation in other cohorts and ethnic groups.
A randomised controlled trial for overweight and obese parents to prevent childhood obesity - Early STOPP (STockholm Obesity Prevention Program)
Tanja Sobko, Viktoria Svensson, Anna Ek, Mirjam Ekstedt, H?kan Karlsson, Elin Johansson, Yingting Cao, Maria Hagstr?mer, Claude Marcus
BMC Public Health , 2011, DOI: 10.1186/1471-2458-11-336
Abstract: This randomized controlled trial (RCT) targets overweight and/or obese parents with infants, recruited from the Child Health Care Centers (CHCC) within the Stockholm area. The intervention starts when infants are one year of age and continues until they are six and is regularly delivered by a trained coach (dietitian, physiotherapist or a nurse). The key aspects of Early STOPP family intervention are based on Swedish recommendations for CHCC, which include advices on healthy food choices and eating patterns, increasing physical activity/reducing sedentary behavior and regulating sleeping patterns.The Early STOPP trial design addresses weaknesses of previous research by recruiting from a well-characterized population, defining a feasible, theory-based intervention and assessing multiple measurements to validate and interpret the program effectiveness. The early years hold promise as a time in which obesity prevention may be most effective. To our knowledge, this longitudinal RCT is the first attempt to demonstrate whether an early, long-term, targeted health promotion program focusing on healthy eating, physical activity/reduced sedentary behaviors and normalizing sleeping patterns could be effective. If proven so, Early STOPP may protect children from the development of overweight and obesity.The protocol for this study is registered with the clinical trials registry clinicaltrials.gov, ID: ES-2010)Overweight is considered to be a global epidemic and the marked increase in childhood obesity is alarming. Preventing the development of obesity in children is therefore a world-wide health priority [1]. Childhood obesity is multi-factorial and has been recognized as heritable [2]. The odds for a child to become obese as an adult increase about threefold if one of the parents is obese and rise tenfold with two obese parents [3]. To which extent this hereditariness is socio-cultural, genetic or epigenetic is unclear [4].Gain in body weight and fat mass is a consequence of
The STK33-Linked SNP rs4929949 Is Associated with Obesity and BMI in Two Independent Cohorts of Swedish and Greek Children
Mathias Rask-Andersen, George Moschonis, George P. Chrousos, Claude Marcus, George V. Dedoussis, Robert Fredriksson, Helgi B. Schi?th
PLOS ONE , 2013, DOI: 10.1371/journal.pone.0071353
Abstract: Recent genome wide association studies (GWAS) have identified a locus on chromosome 11p15.5, closely associated with serine/threonine kinase 33 (STK33), to be associated with body mass. STK33, a relatively understudied protein, has been linked to KRAS mutation-driven cancers and explored as a potential antineoplastic drug target. The strongest association with body mass observed at this loci in GWAS was rs4929949, a single nucleotide polymorphism located within intron 1 of the gene encoding STK33. The functional implications of rs4929949 or related variants have not been explored as of yet. We have genotyped rs4929949 in two cohorts, an obesity case-control cohort of 991 Swedish children, and a cross-sectional cohort of 2308 Greek school children. We found that the minor allele of rs4929949 was associated with obesity in the cohort of Swedish children and adolescents (OR = 1.199 (95%CI: 1.002–1.434), p = 0.047), and with body mass in the cross-sectional cohort of Greek children (β = 0.08147 (95% CI: 0.1345–0.1618), p = 0.021). We observe the effects of rs4929949 on body mass to be detectable already at adolescence. Subsequent analysis did not detect any association of rs4929949 to phenotypic measurements describing body adiposity or to metabolic factors such as insulin levels, triglycerides and insulin resistance (HOMA).
Thermodynamic cost of creating correlations
Marcus Huber,Martí Perarnau-Llobet,Karen V. Hovhannisyan,Paul Skrzypczyk,Claude Kl?ckl,Nicolas Brunner,Antonio Acín
Physics , 2014, DOI: 10.1088/1367-2630/17/6/065008
Abstract: We investigate the fundamental limitations imposed by thermodynamics for creating correlations. Considering a collection of initially uncorrelated thermal quantum systems, we ask how much classical and quantum correlations can be obtained via a cyclic Hamiltonian process. We derive bounds on both the mutual information and entanglement of formation, as a function of the temperature of the systems and the available energy. While for a finite number of systems there is a maximal temperature allowing for the creation of entanglement, we show that genuine multipartite entanglement---the strongest form of entanglement in multipartite systems---can be created at any temperature when sufficiently many systems are considered. This approach may find applications, e.g. in quantum information processing, for physical platforms in which thermodynamic considerations cannot be ignored.
The “3 Genomic Numbers” Discovery: How Our Genome Single-Stranded DNA Sequence Is “Self-Designed” as a Numerical Whole  [PDF]
Jean-Claude Perez
Applied Mathematics (AM) , 2013, DOI: 10.4236/am.2013.410A2004
Abstract:

This article proves the existence of a hyper-precise global numerical meta-architecture unifying, structuring, binding and controlling the billion triplet codons constituting the sequence of single-stranded DNA of the entire human genome. Beyond the evolution and erratic mutations like transposons within the genome, it’s as if the memory of a fossil genome with multiple symmetries persists. This recalls the intermingling of information characterizing the fractal universe of chaos theory. The result leads to a balanced and perfect tuning between the masses of the two strands of the huge DNA molecule that constitute our genome. We show here how codon populations forming the single-stranded DNA sequences can constitute a critical approach to the understanding of junk DNA function. Then, we suggest revisiting certain methods published in our 2009 book “Codex Biogenesis”. In fact, we demonstrate here how the universal genetic code table is a powerful analytical filter to characterize single-stranded DNA sequences constituting chromosomes and genomes. We can then show that any genomic DNA sequence is featured by three numbers, which characterize it and its 64 codon populations with correlations greater than 99%. The number “1” is common to all sequences, expressing the second law of Chargaff. The other 2 numbers are related to each specific DNA sequence case characterizing life species. For example, the entire human genome is characterized by three remarkable numbers 1, 2, and Phi = 1.618 the golden ratio. Associated with each of these three numbers, we can match three axes of symmetry, then “imagine” a kind of hyperspace formed by these codon populations. Then we revisit the value (3-Phi)/2 which is probably universal and common to both the scale of quarks and atomic levels, balancing and tuning the whole human genome codon population. Finally, we demonstrate a new kind of duality between “form and substance” overlapping the whole human genome: we will show that—simultaneously with the duality between genes and junk DNA—there is a second layer of embedded hidden structure overlapping all the DNA of the whole human genome, dividing it into a second type of duality information/redundancy involving golden ratio proportions.

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