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Search Results: 1 - 10 of 2644 matches for " Cerebral contusion "
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Contus?o cerebral em vítimas fatais de acidente de transito: frequência e associa??o com outras les?es cranioencefálicas
PITTELLA, JOSé EYMARD HOMEM;GUSM?O, SEBASTI?O SILVA;
Arquivos de Neuro-Psiquiatria , 1999, DOI: 10.1590/S0004-282X1999000500018
Abstract: a morphological study, macro and microscopical, was made of brain lesions in 120 victims of fatal road traffic accidents. contusions of the brain were identified in 67 (55.8%) of the patients. the contusions especially affected the orbital surfaces of the frontal lobes (27.8%) and the temporal poles (19.8%). the recent brain contusion was associated with a skull fracture in 70.1% of the cases and with a diffuse axonal injury in 89.5%. these can be explained by the association of contact and inertia from the road traffic accidents. old contusions were identified in six patients (5.0%).
Modelos experimentales de traumatismo craneoencefálico
Prieto,R.; Gutiérrez-González,R.; Pascual,J.M.; Roda,J.M.; Cerdán,S.; Matias-Guiu,J.; Barcia,J.A.;
Neurocirugía , 2009, DOI: 10.4321/S1130-14732009000300001
Abstract: aim. to provide a summary of the different experimental models of traumatic brain injury (tbi) designed under both in vivo and in vitro conditions. a comprehensible review of the specific types of brain lesions induced, as well as the technical details to reproduce each model at the laboratory is given. development. outcome of patients suffering from a tbi has significantly improved with the rapid application of vital supporting measures in addition to a strict control of blood and intracranial pressure at the intensive care units. however no specific treatment for post-traumatic brain lesions has proven as efficacious in the clinical settings. a deeper knowlegde of the physiopathological events associated with tbi is necessary for the development of new specific therapies. due to the heterogeneity of the human tbi, each experimental model has been designed to reproduce a different type of brain lesion. experimental tbi models allow the study of the dynamic evolution of brain injuries under controlled conditions. usefulness of experimental models is limited by their reliability and reproducibility among different researchers. small rodents have been the preferred animals to reproduce tbi injuries, mainly due to the similar cerebral physiology shared by these animals and the human beings. conclusion. the use of experimental models of tbi is the most appropiate tool to study the mechanisms underlying this type of injury. however their simplicity precludes an exact reproduction of the heterogeneous cerebral damage observed in clinical settings. this could be the main reason for the discrepancies observed in the therapeutic effects of treatments between experimental and clinical studies.
Evolución electro-clínica a distancia de la contusión cerebral traumática en el ni o: Riesgo de la secuela epiléptica
Nelly Chiofalo S,Antonio Fuentes E,Luciano Basauri T,Juan Madsen L
Revista chilena de pediatría , 1980,
Abstract:
Neurosurgical treatment of posttraumatic intracerebral haematomas
?urovi? B.,Jovanovi? V.,Tasi? Goran,Ka?ar Aleksandra
Acta Chirurgica Iugoslavica , 2003, DOI: 10.2298/aci0302071d
Abstract: From January 1st to August 31st 2002 yr., Neurosurgical department of the Trauma Center, Clinical Center of Serbia, has operated 43 patients with posttraumatic intracerebral haemathoma (PTIH). From that number, 9 patients survived and 34 died. Only 4 patients with acute PTIH were in terminal state of incarceration and in spite they were operated immediately, all died. Other 39 patients have delayed PTIH where secondary CT cerebral scans showed the development of posttraumatic intracerebral haematoma that has not been verified at the incipient scanner. Indication for repeated CT scan was found for 19 patients for their focal or general neurological deterioration. 20 patients had no delayed neurological disturbances. Survivers were younger, in lower grade of coma and were mostly with temporal localization of haemathoma.
Missile injuries of the brain: Results of less aggressive surgery
Singh P
Neurology India , 2003,
Abstract:
Modelos experimentales de traumatismo craneoencefálico Experimental models of traumatic brain injury
R. Prieto,R. Gutiérrez-González,J.M. Pascual,J.M. Roda
Neurocirugía , 2009,
Abstract: Objetivo. El objetivo de este trabajo es proporcionar una revisión de los diversos modelos experimentales de traumatismo craneoencefálico (TCE) que se han desarrollado para la investigación del da o cerebral traumático tanto en condiciones in vivo como in vitro, así como detallar los principales conocimientos fisiopatológicos obtenidos a partir de su aplicación. Se expone de forma sintética tanto el tipo de lesión cerebral traumática que cada modelo reproduce como los detalles técnicos necesarios para su utilización por investigadores en el campo del trauma cerebral. Desarrollo. El pronóstico de los pacientes que han sufrido un TCE ha mejorado gracias a las medidas iniciales de estabilización hemodinámica y control de la vía aérea, pero no existe todavía ningún tratamiento específico y eficaz para detener o limitar las lesiones cerebrales causadas por el traumatismo, exceptuando las medidas de control de la presión arterial y la presión intracraneal. Entender la fisiopatología del TCE es el paso básico y fundamental para desarrollar posibles abordajes terapéuticos con aplicación clínica. El da o cerebral traumático en humanos es una patología heterogénea y muy compleja. Por ello, cada modelo experimental se ha desarrollado con el objetivo de reproducir un tipo concreto de las diferentes lesiones cerebrales observadas en pacientes tras un TCE. El uso de estos modelos ha permitido ampliar el conocimiento sobre la fisiopatología del da o cerebral traumático, incluyendo las alteraciones inducidas a nivel celular y molecular. Conclusión. Los modelos experimentales suponen actualmente la mejor herramienta para el estudio de los mecanismos subyacentes a las lesiones cerebrales traumáticas, pero su simplicidad y por lo tanto su incapacidad de reproducir exactamente el da o heterogéneo observado en la práctica clínica puede ser uno de los motivos que explique la discrepancia en la respuesta terapéutica entre los estudios experimentales y clínicos. Aim. To provide a summary of the different experimental models of traumatic brain injury (TBI) designed under both in vivo and in vitro conditions. A comprehensible review of the specific types of brain lesions induced, as well as the technical details to reproduce each model at the laboratory is given. Development. Outcome of patients suffering from a TBI has significantly improved with the rapid application of vital supporting measures in addition to a strict control of blood and intracranial pressure at the intensive care units. However no specific treatment for post-traumatic brain lesions has proven as efficacious
Trombose carotídea após traumatismo contuso
Bez, Leonardo Ghizoni;Drumond, Domingos André Fernandes;
Revista do Colégio Brasileiro de Cirurgi?es , 2001, DOI: 10.1590/S0100-69912001000300016
Abstract: the authors report a case of thrombosis of common and internal carotid arteries caused by blunt trauma. they emphasize the rarity of this lesion and the variability of clinical presentation. different forms of treatment are analysed, such as the use of anticoagulation and surgery. treatment needs to be individualized for each patient.
Utilidad de la TAC secuencial y la monitorización de la presión intracraneal para detectar nuevo efecto masa intracraneal en pacientes con traumatismo craneal grave y lesión inicial Tipo I-II Value of serial CT scanning and intracranial pressure monitoring for detecting new intracranial mass effect in severe head injury patients showing lesions type I-II in the initial CT scan
R.D. Lobato,J.F. Alen,A. Perez-Nu?ez,R. Alday
Neurocirugía , 2005,
Abstract: Objetivo. Analizar los cambios en la patología y presión intracraneal (PIC) durante el periodo agudo postraumático en una serie de pacientes con trauma craneal grave y lesiones Tipos I-II en la TAC inicial (clasificación del Traumatic Coma Data Bank) con el objetivo de dise ar la pauta mas adecuada de uso de TAC secuencial y monitorización de la PIC para detectar nuevo efecto masa intracraneal y tratar así de mejorar la evolución final de pacientes. Material y métodos. Se analiza una serie de 56 pacientes (edades = 15-80 a os) admitidos consecutivamente en un periodo de dos a os que fueron sometidos a TAC inicial < 24 horas tras el impacto, (intervalo medio = 150 minutos), TACs control en los primeros días del curso, y monitorización de la PIC. Se recogieron diferentes variables epidemiológicas, clínicas, radiológicas y se consideró como variable dependiente el desarrollo de deterioro definido como elevación mantenida de la PIC por encima de 20 mmHg que requiriera tratamiento agresivo médico y/o quirúrgico. Mediante análisis bi y multivariante se determinaron las correlaciones entre las diferentes variables y la aparición de deterioro. Para estimar la afectación neurológica y el resultado final se emplearon las escalas de coma y evolución de Glasgow, respectivamente. Resultados. El "score " medio en la serie fue de 5, y 37% de los pacientes tuvieron cambios pupilares, 52,3% hipotensión-hipoxemia, 16.1% anemia peritraumáticas y 12,3% alteraciones de la coagulación. 50% de los pacientes mostraron petequias en sustancia blanca y/o tronco cerebral en la TAC inicial, 66% HSA, 40% HIV, 39,3% contusión y 21,4% hematomas extraaxiales. 57,1% de los pacientes mostraron cambios en la TAC de control consistentes en nueva contusión en 26,8% de los casos, crecimiento de contusión previa en 68,2%, crecimiento de hematoma previo en 10,7% y swelling cerebral generalizado en 10,7%. 64% de los pacientes experimentaron una evolución final favorable y 35,7% desfavorable. 27 pacientes (48,9%) desarrollaron deterioro PIC, de los que 21 (37,5%) presentaron cambios concurrentes en la TAC, y 6 (10,7%) no los mostraron. Los restantes 29 (51,7%) pacientes no presentaron deterioro PIC, aunque 11 (19,6%) de ellos mostraron cambio TAC. La edad, el "score", la presencia de hipotension-hipoxemia peritraumáticas y los trastornos de la coagulación no se correlacionaron con riesgo de deterioro. Por el contrario, la presencia de contusión inicial (p=0,01) y el cambio TAC (en forma de desarrollo de swelling cerebral generalizado, p=0,003) se correlacionaron con la aparición de deterioro; a s
Les?es traumáticas do parênquima pulmonar: aspectos na tomografia computadorizada
Melo, Alessandro Severo Alves de;Moreira, Luiza Beatriz Melo;Marchiori, Edson;
Radiologia Brasileira , 2003, DOI: 10.1590/S0100-39842003000300004
Abstract: traumatic lesions of the lung are common findings in patients with thoracic trauma. these lesions are increasingly diagnosed using computed tomography, mostly due to the fast acquisition time helical techniques that allow evaluation of critically ill patients and an efficient therapeutic management. the authors studied 150 patients with thoracic trauma submitted to computed tomography that demonstrated lung contusions, atelectasies, lacerations and hematomas. lung contusions were the most frequent lesions appearing as consolidation or ground-glass attenuation areas. atelectasies, in subsegmentar and compressive patterns, were the second most common lesions observed. lacerations appeared as consolidations with air or liquid level. lung hematomas, characterized by round opacities, were the most rare lung lesions seen in only five cases. in this study, blunt thoracic trauma accounted for the majority of 120 cases whereas penetrating trauma occurred in 30 cases. the causes of blunt trauma in decrescent order of frequency were motor vehicle accidents, pedestrian hit by car, falls, motorcycle accidents and trashing. penetrating traumas were caused by bullets or knives.
Epidemiologia do trauma facial
Wulkan, Marcelo;Parreira Jr, José Gustavo;Botter, Denise Aparecida;
Revista da Associa??o Médica Brasileira , 2005, DOI: 10.1590/S0104-42302005000500022
Abstract: objectives: this study aims to determine the incidence, etiology, severity of facial trauma and associated injuries enabling a greater understanding of its range and magnitude methods: a hundred and sixty four patients were selected with some degree of facial trauma regardless of gender, age and skin color. data were analyzed by the pearson x2 statistical method. results: a male predominance was observed (78%) and its peak age was between 20 and 39 years. the major cause was interpersonal violence (48.1%), followed by fall (26.2%), run overs 6.4%), sports (5.4%), car accidents (4.2%), motorcycle accidents (3.1%), non-fall impacts (2.4%), occupational injuries (1.8%), gunshot wounds (1.2%), unspecific (1.2%). contusion is the most common injury (23.8%), followed by fractures of the mandible (21.9%), le fort/pan facial/complex (17.8%), nasal bones (11.6%), zygoma (10.3%), tooth (9.1%), orbit (4.9%) and maxilla (0.6%). associated injuries epidemiologia do trauma facial occurred mostly in run overs, but also because of car accident, fall and interpersonal violence. conclusions: the causes of facial trauma are directly related to the age and type of lesion. no evidence was found that the causes were related to gender or severity of the lesion.
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