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Helicobacter pylori y coriorretinopatía serosa central Helicobacter pylori and central Selous chorioretinopathy  [cached]
R Abreu,J Nadal,P Abreu
Archivos de la Sociedad Espa?ola de Oftalmología , 2008,
Abstract:
Incidence of Helicobacter pylori in central serous chorioretinopathy: a case control study  [PDF]
Mostafa Feghhi,Eskandar Hajiani,Gholamreza Khataminia
Jundishapur Journal of Microbiology , 2008,
Abstract: Helicobacter pylori infection is one of the most common infections worldwide. Central serous chorioretinopathy (CSCR) is a serous macular detachment that usually affects young people. The aetiopathogenesis of the disease is still not completely understood. Recently, an interesting association has been observed between this disease and the H. pylori infection. This study was conducted to investigate a possible association between H. pylori infection and CSCR. A prospective study was performed and we evaluated a total of 54 CSCR patients (48 males and 6 females, median age 35.7 years), and a control group of 59 patients (25 women, 34 males; mean age 42.6±11 years) who referred to gastroenterology department of Imam Hospital, Ahvaz Jundishapur University of Medical Sciences, were studied. Central serous chorioretinopathy was diagnosed on the basis of findings in ophthalmic examinations and confirmed by fluorescein angiogram. All patients underwent a 13C-urea breath test (UBT) and serum IgG anti-bodies to H. pylori by enzyme-linked immunosorbent assay technique to detect H. pylori infection. Patients were defined as H. pylori infected if both tests were positive. The mean duration of symptoms before diagnosis was 10.5±4.5 days. Overall no statistically significant difference was found between left and right eyes, bilaterally was in 5 patients (9.2%). The incidence of H. pylori infection was 68.5% in CSCR patients and 65% in control subjects (p=0.64). These results indicate that the prevalence of H. pylori infection is not higher in patients with CSCR than in controls. Further large studies will be required to determine the role of H. pylori infection in patients with CSCR.
Helicobacter pylori infection is identified as a cardiovascular risk factor in Central Africans
Longo-Mbenza B, Nsenga JN, Mokondjimobe E, Gombet T, Assori IN, Ibara JR, Ellenga-Mbolla B, Vangu DN, Fuele SM
Vascular Health and Risk Management , 2012, DOI: http://dx.doi.org/10.2147/VHRM.S28680
Abstract: obacter pylori infection is identified as a cardiovascular risk factor in Central Africans Original Research (1368) Total Article Views Authors: Longo-Mbenza B, Nsenga JN, Mokondjimobe E, Gombet T, Assori IN, Ibara JR, Ellenga-Mbolla B, Vangu DN, Fuele SM Published Date August 2012 Volume 2012:8 Pages 455 - 461 DOI: http://dx.doi.org/10.2147/VHRM.S28680 Received: 29 November 2011 Accepted: 25 January 2012 Published: 15 August 2012 Benjamin Longo-Mbenza,1 Jacqueline Nkondi Nsenga,2 Etienne Mokondjimobe,3 Thierry Gombet,3 Itoua Ngaporo Assori,3 Jean Rosaire Ibara,3 Bertrand Ellenga-Mbolla,3 Dieudonné Ngoma Vangu,4 Simon Mbungu Fuele4 1Faculty of Health Sciences, Walter Sisulu University, Mthatha, South Africa; 2Division of Gastroenterology, University of Kinshasa, Kinshasa, Democratic Republic of the Congo; 3Faculty of Health Sciences, University of Marien Ngouabi, Brazzaville, Democratic Republic of the Congo; 4Biostatistics Unit, Lomo Medical Center, Limete, Kinshasa, Democratic Republic of the Congo Background: Helicobacter pylori is now incriminated in the pathogenesis of atherosclerosis. Objective: To examine the importance of H. pylori infection as a cardiovascular disease (CVD) risk factor. Methods: Two hundred five patients (128 with H. pylori infection [HP-seropositive] and 77 without) had a baseline assessment for other potential CVD risk factors and were followed prospectively for 10 years (1999–2008). They were assessed on a monthly basis for the outcomes of carotid plaque, angina pectoris, myocardial infarction, and stroke. In the HP-seropositive group, male sex and quartile 4 for IgG anti-H. pylori antibodies (anti-HP Ab) were correlated with traditional CVD risk factors, stroke, myocardial infarction, and angina pectoris. Results: At the baseline assessment, the levels of carotid intima-media thickness, blood fibrinogen, total cholesterol, fasting plasma glucose, and uric acid were higher in H. pylori-infected patients than in the uninfected group. Serum HDL-cholesterol was significantly lower in the HP-seropositive group. Men had higher levels of IgG anti-HP Ab, waist circumference, blood pressure, uric acid, and total cholesterol than women. Within the HP-seropositive group, individuals in quartile 4 for IgG anti-HP Ab had higher rates of elevated fibrinogen, diabetes mellitus, low high-density lipoprotein cholesterol, arterial hypertension, and high total cholesterol than those in quartile 1. After adjusting for traditional CVD risk factors, H. pylori infection was the only independent predictor of incident carotid plaque (multivariate odds ratio [OR] = 2.3, 95% confidence interval [CI]: 1.2–7.2; P < 0.0001) and incident acute stroke (multivariate OR = 3.6, 95% CI: 1.4–8.2; P < 0.0001). Within the HP-seropositive group and after adjusting for traditional CVD risk factors, male sex was the only independent predictor of incident angina pectoris (multivariate OR = 3.5, 95% CI: 1.6–16; P < 0.0001), incident acute stroke (multivariate
Helicobacter pylori infection is identified as a cardiovascular risk factor in Central Africans  [cached]
Longo-Mbenza B,Nsenga JN,Mokondjimobe E,Gombet T
Vascular Health and Risk Management , 2012,
Abstract: Benjamin Longo-Mbenza,1 Jacqueline Nkondi Nsenga,2 Etienne Mokondjimobe,3 Thierry Gombet,3 Itoua Ngaporo Assori,3 Jean Rosaire Ibara,3 Bertrand Ellenga-Mbolla,3 Dieudonné Ngoma Vangu,4 Simon Mbungu Fuele41Faculty of Health Sciences, Walter Sisulu University, Mthatha, South Africa; 2Division of Gastroenterology, University of Kinshasa, Kinshasa, Democratic Republic of the Congo; 3Faculty of Health Sciences, University of Marien Ngouabi, Brazzaville, Democratic Republic of the Congo; 4Biostatistics Unit, Lomo Medical Center, Limete, Kinshasa, Democratic Republic of the CongoBackground: Helicobacter pylori is now incriminated in the pathogenesis of atherosclerosis.Objective: To examine the importance of H. pylori infection as a cardiovascular disease (CVD) risk factor.Methods: Two hundred five patients (128 with H. pylori infection [HP-seropositive] and 77 without) had a baseline assessment for other potential CVD risk factors and were followed prospectively for 10 years (1999–2008). They were assessed on a monthly basis for the outcomes of carotid plaque, angina pectoris, myocardial infarction, and stroke. In the HP-seropositive group, male sex and quartile 4 for IgG anti-H. pylori antibodies (anti-HP Ab) were correlated with traditional CVD risk factors, stroke, myocardial infarction, and angina pectoris.Results: At the baseline assessment, the levels of carotid intima-media thickness, blood fibrinogen, total cholesterol, fasting plasma glucose, and uric acid were higher in H. pylori-infected patients than in the uninfected group. Serum HDL-cholesterol was significantly lower in the HP-seropositive group. Men had higher levels of IgG anti-HP Ab, waist circumference, blood pressure, uric acid, and total cholesterol than women. Within the HP-seropositive group, individuals in quartile 4 for IgG anti-HP Ab had higher rates of elevated fibrinogen, diabetes mellitus, low high-density lipoprotein cholesterol, arterial hypertension, and high total cholesterol than those in quartile 1. After adjusting for traditional CVD risk factors, H. pylori infection was the only independent predictor of incident carotid plaque (multivariate odds ratio [OR] = 2.3, 95% confidence interval [CI]: 1.2–7.2; P < 0.0001) and incident acute stroke (multivariate OR = 3.6, 95% CI: 1.4–8.2; P < 0.0001). Within the HP-seropositive group and after adjusting for traditional CVD risk factors, male sex was the only independent predictor of incident angina pectoris (multivariate OR = 3.5, 95% CI: 1.6–16; P < 0.0001), incident acute stroke (multivariate OR = 3.2, 95% CI: 1.4–28; P < 0.0001),
Coroidoretinopatia central serosa
Serracarbassa Pedro Duraes
Arquivos Brasileiros de Oftalmologia , 2002,
Abstract: O autor descreve os achados clínicos e novos métodos diagnósticos da coroidoretinopatia central serosa. Discute os estudos experimentais e clínicos relacionados à patogênese da doen a e apresenta as alternativas de tratamento e prognóstico da coroidoretinopatia central serosa, baseado em levantamento bibliográfico.
Immunity and Helicobacter pylori  [cached]
Paul Harris
Medwave , 2011,
Abstract: The bacteria called Helicobacter pylori arrived to the American continent 12,000 years ago (1), reaching South America roughly 5,400-4,600 years AC according to research by Pelayo Correa, a Colombian pathologist who found Helicobacter in stool next to Chinchorro mummies in the North of Arica close to the Pacific Ocean. In 2005, Barry Marshall was awarded the Nobel Prize for his studies on Helicobacter pylori together with Robin Warren.
Coroidoretinopatia central serosa
Serracarbassa, Pedro Duraes;
Arquivos Brasileiros de Oftalmologia , 2002, DOI: 10.1590/S0004-27492002000300020
Abstract: the author describes the clinical features and new diagnostic methods of central serous chorioretinopathy. he discusses experimental and clinical studies related to the pathogenesis of the disease and presents therapy choices and prognosis of central serous chorioretinopathy, based on reference survey.
Behcet’s Disease and Helicobacter Pylori  [PDF]
Ramazan Dan??,Kadim Bayan,?erif Y?lmaz,Abdullah Alt?nta?
Dicle Medical Journal , 2004,
Abstract: The aim of this study is to investigate the relation between Beh et’s disease (BD) and Helicobacter pylori infection. The patients fulfilled the criteria of the International Study Group for Beh et’s disease. Helicobacter pylori infection was diagnosed by the rapid urease test and esophagogastroduodenoscopic examination performed to BD patients. Fourteen of the 22 patients with BD were Helicobacter pylori positive with rapid urease test. All patients with ophthalmic involvement were Helicobacter pylori positive with rapid urease test.
DIAGNOSIS OF HELICOBACTER PYLORI INFECTION  [cached]
Vladan N. Petrovi?
Acta Facultatis Medicae Naissensis , 2003,
Abstract: Helicobacter pylori is the most widen infection on the World. It is importrant factor in the genesis of ulcer disease, gastric carcinoma, gastric lymphoma, as a numerous extragastric disease. Diagnosis is made by invasive and non-invasive tests. Maastricht Consensus Report (2000) and First Consensus of Yugoslav Gastroenterologic Association recommend group of patients and methods for detection of Helicobacter pylori infection.
HISTORY OF HELICOBACTER PYLORI INFECTION  [cached]
Tomica Milosavljevi?,Aleksandar Nagorni
Acta Facultatis Medicae Naissensis , 2003,
Abstract: Helicobacter pylori was identified in 1979, but cultivated in Australia in 1982. Warren and Marshall were responsible for discovery. But, in the previous one hundred years, a lot of scientists tried to prove hipothesis about possible association between peptic ulcer disease and spiral microorganism. Numerous authors were objectivated H. pylori on some way. Consensus Conferences in Maastricht in 1996 and 2000 and First Consensus of Yugoslav Gastroenterologic Association gave us recommendation and guideline for diagnostics and treatment of H. pylori infection.
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